Gregory B. Waypa

O(2) sensing in hypoxic pulmonary vasoconstriction: the mitochondrial door re-opens.

Mitochondrial reactive oxygen species trigger calcium increases during hypoxia in pulmonary arterial myocytes.

Hypoxic pulmonary vasoconstriction: redox events in oxygen sensing.

Role for mitochondrial reactive oxygen species in hypoxic pulmonary vasoconstriction.

Increases in mitochondrial reactive oxygen species trigger hypoxia-induced calcium responses in pulmonary artery smooth muscle cells.

Oxidant stress during simulated ischemia primes cardiomyocytes for cell death during reperfusion.

Oxygen sensing in hypoxic pulmonary vasoconstriction: using new tools to answer an age-old question.

Prolonged hypoxia increases ROS signaling and RhoA activation in pulmonary artery smooth muscle and endothelial cells.

Hypoxia increases ROS signaling and cytosolic Ca(2+) in pulmonary artery smooth muscle cells of mouse lungs slices.

Hypoxia triggers subcellular compartmental redox signaling in vascular smooth muscle cells.

Grape seed proanthocyanidins ameliorate Doxorubicin-induced cardiotoxicity.

Hypoxia-induced changes in pulmonary and systemic vascular resistance: where is the O2 sensor?

Mitochondrial oxidant stress triggers cell death in simulated ischemia-reperfusion.

Hypoxia triggers AMPK activation through reactive oxygen species-mediated activation of calcium release-activated calcium channels.

Superoxide generated at mitochondrial complex III triggers acute responses to hypoxia in the pulmonary circulation.

Peroxiredoxin-5 targeted to the mitochondrial intermembrane space attenuates hypoxia-induced reactive oxygen species signaling.

Sirt3 Deficiency Does not Augment Hypoxia-Induced Pulmonary Hypertension.