Martine Dunnwald

Mouse epidermal stem cells proceed through the cell cycle.

Leprdb diabetic mouse bone marrow cells inhibit skin wound vascularization but promote wound healing.

Epidermal cells accelerate the restoration of the blood flow in diabetic ischemic limbs.

Obese diabetic mouse environment differentially affects primitive and monocytic endothelial cell progenitors.

Abnormal skin, limb and craniofacial morphogenesis in mice deficient for interferon regulatory factor 6 (Irf6).

Murine epidermal side population possesses unique angiogenic properties.

A genome-wide association study of cleft lip with and without cleft palate identifies risk variants near MAFB and ABCA4.

Lin- cells mediate tissue repair by regulating MCP-1/CCL-2.

Wound complications after cleft repair in children with Van der Woude syndrome.

Antioxidant proteins and reactive oxygen species are decreased in a murine epidermal side population with stem cell-like characteristics.

Genomic strategy identifies a missense mutation in WD-repeat domain 65 (WDR65) in an individual with Van der Woude syndrome.

Interferon regulatory factor 6 is necessary, but not sufficient, for keratinocyte differentiation.

MCS9.7 enhancer activity is highly, but not completely, associated with expression of Irf6 and p63.

Interferon regulatory factor 6 promotes differentiation of the periderm by activating expression of Grainyhead-like 3.

Expression and mutation analyses implicate ARHGAP29 as the etiologic gene for the cleft lip with or without cleft palate locus identified by genome-wide association on chromosome 1p22.

Transforming growth factor Beta 3 is required for excisional wound repair in vivo.

A Novel non-sense Mutation in Keratin 10 Causes a Familial Case of Recessive Epidermolytic Ichthyosis.

Haploinsufficiency of interferon regulatory factor 6 alters brain morphology in the mouse.

Dominant mutations in GRHL3 cause Van der Woude Syndrome and disrupt oral periderm development.

Digital imaging analysis to assess scar phenotype.

Interferon regulatory factor 6 regulates keratinocyte migration.

Palatogenesis and cutaneous repair: A two-headed coin.

Expanding the genetic and phenotypic spectrum of popliteal pterygium disorders.

Irf6 directly regulates Klf17 in zebrafish periderm and Klf4 in murine oral epithelium, and dominant-negative KLF4 variants are present in patients with cleft lip and palate.

Interferon Regulatory Factor 6 Has a Protective Role in the Host Response to Endotoxic Shock.

Interferon Regulatory Factor 6 Controls Proliferation of Keratinocytes From Children With Van der Woude Syndrome.

Exome sequencing provides additional evidence for the involvement of ARHGAP29 in Mendelian orofacial clefting and extends the phenotypic spectrum to isolated cleft palate.

A Case Study of Malrotated Kidneys with Asymmetric Multiple Renal Arteries, Variant Venous Drainage, and Unilateral Ureteral Duplication.

Interferon regulatory factor 6 is required for proper wound healing in vivo.

Non-random distribution of deleterious mutations in the DNA and protein-binding domains of IRF6 are associated with Van Der Woude syndrome.