Michael Petrascheck

Organ polarity in Arabidopsis. NOZZLE physically interacts with members of the YABBY family.

Quenching accumulation of toxic galactose-1-phosphate as a system to select disruption of protein-protein interactions in vivo.

DNA looping induced by a transcriptional enhancer in vivo.

An antidepressant that extends lifespan in adult Caenorhabditis elegans.

A high-throughput screen for chemicals that increase the lifespan of Caenorhabditis elegans.

A pharmacological network for lifespan extension in Caenorhabditis elegans.

The metabolite α-ketoglutarate extends lifespan by inhibiting ATP synthase and TOR.

High-throughput small-molecule screening in Caenorhabditis elegans.

Pharmacological classes that extend lifespan of Caenorhabditis elegans.

Measuring Food Intake and Nutrient Absorption in Caenorhabditis elegans.

Atypical antidepressants extend lifespan of Caenorhabditis elegans by activation of a non-cell-autonomous stress response.

Suppression of transcriptional drift extends C. elegans lifespan by postponing the onset of mortality.

C. elegans S6K Mutants Require a Creatine-Kinase-like Effector for Lifespan Extension.

Proton Pump Inhibitors Accelerate Endothelial Senescence.

Metabolic drift in the aging brain.

C. elegans as Model for Drug Discovery.

The DrugAge database of aging-related drugs.

The mitochondrial ATP synthase is a shared drug target for aging and dementia.

Translation attenuation by minocycline enhances longevity and proteostasis in old post-stress-responsive organisms.

A phenotypic Caenorhabditis elegans screen identifies a selective suppressor of antipsychotic-induced hyperphagia.

Loss of genomic integrity induced by lysosphingolipid imbalance drives ageing in the heart.

Pharmacological convergence reveals a lipid pathway that regulates C. elegans lifespan.

Elevating acetyl-CoA levels reduces aspects of brain aging.

Design and Analysis of Pharmacological Studies in Aging.

The aging transcriptome: read between the lines.

A chemical biology approach to identifying molecular pathways associated with aging.

Rapamycin-mediated mouse lifespan extension: Late-life dosage regimes with sex-specific effects.

Adipocytes control food intake and weight regain via Vacuolar-type H ATPase.

Targeting Clic1 for the treatment of obesity: A novel therapeutic strategy to reduce food intake and body weight.