Michel Favre

Mutations in two adjacent novel genes are associated with epidermodysplasia verruciformis.

Severe cutaneous papillomavirus disease after haemopoietic stem-cell transplantation in patients with severe combined immune deficiency caused by common gammac cytokine receptor subunit or JAK-3 deficiency.

Genomic organization of amplified MYC genes suggests distinct mechanisms of amplification in tumorigenesis.

Human papillomavirus type 5 E6 oncoprotein represses the transforming growth factor beta signaling pathway by binding to SMAD3.

[Genetics and business: in defence of free speech!].

Regulation of cellular zinc balance as a potential mechanism of EVER-mediated protection against pathogenesis by cutaneous oncogenic human papillomaviruses.

[Are oncogenic cutaneous HPVs defective commensals?].

Role of Zn2+ ions in host-virus interactions.

Serological relationship between cutaneous human papillomavirus types 5, 8 and 92.

The EVER proteins as a natural barrier against papillomaviruses: a new insight into the pathogenesis of human papillomavirus infections.

Effects of HPV-16 E5, E6 and E7 proteins on survival, adhesion, migration and invasion of trophoblastic cells.

Seroreactivity of 38 human papillomavirus types in epidermodysplasia verruciformis patients, relatives, and controls.

[Investigation on HPV DNA detection and genotyping practices used in French laboratories in 2009].

Keratinocyte sensitization to tumour necrosis factor-induced nuclear factor kappa B activation by the E2 regulatory protein of human papillomaviruses.

Interleukin-36-receptor antagonist deficiency and generalized pustular psoriasis.

Benchmarking a luciferase complementation assay for detecting protein complexes.

Large scale genotype comparison of human papillomavirus E2-host interaction networks provides new insights for e2 molecular functions.

EVER proteins, key elements of the natural anti-human papillomavirus barrier, are regulated upon T-cell activation.

Comparative analysis of virus-host interactomes with a mammalian high-throughput protein complementation assay based on Gaussia princeps luciferase.

Exploring individual HPV coinfections is essential to predict HPV-vaccination impact on genotype distribution: a model-based approach.