Monika Majewska-Szczepanik

Down-regulation of macrophage immune activity by natural CD8+ regulatory T cells.

IRAK-M deficiency promotes the development of type 1 diabetes in NOD mice.

Epicutaneous immunization with TNP-Ig and Zymosan induces TCRαβ+ CD4+ contrasuppressor cells that reverse skin-induced suppression via IL-17A.

Epicutaneous Immunization with Collagen Induces TCRαβ Suppressor T Cells That Inhibit Collagen-Induced Arthritis.

Inhibitory effect of antidepressant drugs on contact hypersensitivity reaction is connected with their suppressive effect on NKT and CD8(+) T cells but not on TCR delta T cells.

Expression of activation-induced cytidine deaminase enhances the clearance of pneumococcal pneumonia: evidence of a subpopulation of protective anti-pneumococcal B1a cells.

A subset of AID-dependent B-1a cells initiates hypersensitivity and pneumococcal pneumonia resistance.

Oral treatment with enrofloxacin early in life promotes Th2-mediated immune response in mice.

Epicutaneous immunization with ovalbumin and CpG induces TH1/TH17 cytokines, which regulate IgE and IgG2a production.

Epicutaneous (EC) immunization with type II collagen (COLL II) induces CD4(+) CD8(+) T suppressor cells that protect from collagen-induced arthritis (CIA).

Transdermal immunotherapy: Past, present and future.

Broad spectrum antibiotic enrofloxacin modulates contact sensitivity through gut microbiota in a murine model.

Corrigendum to "Oral treatment with enrofloxacin early in life promotes Th2-mediated immune response in mice" [Pharmacol. Rep. 68 (2016) 44-50].

Corrigendum to "Inhibition of 2,4-dinitrofluorobenzene-induced contact hypersensitivity reaction by antidepressant drugs" [Pharmacol. Rep. 65 (2013) 1237-1246].

Regulation of contact sensitivity in non-obese diabetic (NOD) mice by innate immunity.

TRIF deficiency protects non-obese diabetic mice from type 1 diabetes by modulating the gut microbiota and dendritic cells.

Antibiotics and autoimmune and allergy diseases: Causative factor or treatment?

Post-operative complications in patients with a significant post-operative decrease in γδT cells.

Oral treatment with enrofloxacin creates anti-inflammatory environment that supports induction of tolerogenic dendritic cells.

Insulin-Reactive T Cells Convert Diabetogenic Insulin-Reactive VH125 B Cells Into Tolerogenic Cells by Reducing Germinal Center T:B Cell Interactions in NOD Mice.

Diet-induced obesity aggravates NK cell-mediated contact hypersensitivity reaction in Rag1 mice.

Obesity aggravates contact hypersensitivity reaction in mice.