Shermali Gunawardena

Disruption of axonal transport by loss of huntingtin or expression of pathogenic polyQ proteins in Drosophila.

Cargo-carrying motor vehicles on the neuronal highway: transport pathways and neurodegenerative disease.

Polyglutamine diseases and transport problems: deadly traffic jams on neuronal highways.

Amyloid precursor protein-induced axonopathies are independent of amyloid-beta peptides.

Kinesin-1 transport reductions enhance human tau hyperphosphorylation, aggregation and neurodegeneration in animal models of tauopathies.

Organically modified silica nanoparticles are biocompatible and can be targeted to neurons in vivo.