Chih-Wen Ni

Shear flow attenuates serum-induced STAT3 activation in endothelial cells.

Activation of PKC-epsilon and ERK1/2 participates in shear-induced endothelial MCP-1 expression that is repressed by nitric oxide.

Interleukin-6-induced JAK2/STAT3 signaling pathway in endothelial cells is suppressed by hemodynamic flow.

Laminar flow attenuates interferon-induced inflammatory responses in endothelial cells.

Angiopoietin-2 stimulates blood flow recovery after femoral artery occlusion by inducing inflammation and arteriogenesis.

Partial carotid ligation is a model of acutely induced disturbed flow, leading to rapid endothelial dysfunction and atherosclerosis.

Intimal cushions and endothelial nuclear elongation around mouse aortic branches and their spatial correspondence with patterns of lipid deposition.

HuR regulates the expression of stress-sensitive genes and mediates inflammatory response in human umbilical vein endothelial cells.

X-linked inhibitor of apoptosis protein controls alpha5-integrin-mediated cell adhesion and migration.

Discovery of novel mechanosensitive genes in vivo using mouse carotid artery endothelium exposed to disturbed flow.

Animal, in vitro, and ex vivo models of flow-dependent atherosclerosis: role of oxidative stress.

MicroRNA-663 upregulated by oscillatory shear stress plays a role in inflammatory response of endothelial cells.