Jean T. Greenberg

The role and regulation of programmed cell death in plant-pathogen interactions.

Identifying type III effectors of plant pathogens and analyzing their interaction with plant cells.

Ceramides modulate programmed cell death in plants.

A key role for ALD1 in activation of local and systemic defenses in Arabidopsis.

The mitochondrion--an organelle commonly involved in programmed cell death in Arabidopsis thaliana.

Degrade or die: a dual function for autophagy in the plant immune response.

Bioinformatics correctly identifies many type III secretion substrates in the plant pathogen Pseudomonas syringae and the biocontrol isolate P. fluorescens SBW25.

Structure-function analysis of the plasma membrane- localized Arabidopsis defense component ACD6.

Evolutionary dynamics of Ralstonia solanacearum.

A J domain virulence effector of Pseudomonas syringae remodels host chloroplasts and suppresses defenses.

A key role for the Arabidopsis WIN3 protein in disease resistance triggered by Pseudomonas syringae that secrete AvrRpt2.

Arabidopsis proteins important for modulating defense responses to Pseudomonas syringae that secrete HopW1-1.

Genetic analysis of acd6-1 reveals complex defense networks and leads to identification of novel defense genes in Arabidopsis.

A conserved cysteine motif is critical for rice ceramide kinase activity and function.

Accelerated cell death 2 suppresses mitochondrial oxidative bursts and modulates cell death in Arabidopsis.

SGT1b is required for HopZ3-mediated suppression of the epiphytic growth of Pseudomonas syringae on N. benthamiana.

Salicylic acid signaling controls the maturation and localization of the arabidopsis defense protein ACCELERATED CELL DEATH6.

HopW1 from Pseudomonas syringae disrupts the actin cytoskeleton to promote virulence in Arabidopsis.

Loss of ceramide kinase in Arabidopsis impairs defenses and promotes ceramide accumulation and mitochondrial H2O2 bursts.

Salicylic acid regulates Arabidopsis microbial pattern receptor kinase levels and signaling.

ALD1 Regulates Basal Immune Components and Early Inducible Defense Responses in Arabidopsis.

Plant pathogenic bacteria target the actin microfilament network involved in the trafficking of disease defense components.

Arabidopsis AZI1 family proteins mediate signal mobilization for systemic defence priming.

Linking pattern recognition and salicylic acid responses in Arabidopsis through ACCELERATED CELL DEATH6 and receptors.

Acetylation of an NB-LRR Plant Immune-Effector Complex Suppresses Immunity.

Editorial: Salicylic Acid Signaling Networks.

Carbon Nanofiber Arrays: A Novel Tool for Microdelivery of Biomolecules to Plants.

A Suite of Receptor-Like Kinases and a Putative Mechano-Sensitive Channel Are Involved in Autoimmunity and Plasma Membrane-Based Defenses in Arabidopsis.

Simple strategies to enhance discovery of acetylation post-translational modifications by quadrupole-orbitrap LC-MS/MS.

PROHIBITIN3 Forms Complexes with ISOCHORISMATE SYNTHASE1 to Regulate Stress-Induced Salicylic Acid Biosynthesis in Arabidopsis.

Underground Azelaic Acid-Conferred Resistance to Pseudomonas syringae in Arabidopsis.

Kinases and protein motifs required for AZI1 plastid localization and trafficking during plant defense induction.

ALD1 accumulation in Arabidopsis epidermal plastids confers local and non-autonomous disease resistance.

An Improved Bioassay to Study Induced Systemic Resistance (ISR) Against Bacterial Pathogens and Insect Pests.

Pseudomonas syringae effector HopZ3 suppresses the bacterial AvrPto1-tomato PTO immune complex via acetylation.

Friend or foe: Hybrid proline-rich proteins determine how plants respond to beneficial and pathogenic microbes.

The TGA Transcription Factors from Clade II Negatively Regulate the Salicylic Acid Accumulation in Arabidopsis.

An efficient and broadly applicable method for transient transformation of plants using vertically aligned carbon nanofiber arrays.

Genetic requirements for infection-specific responses in conferring disease resistance in .