Vasile Pavlov

Donor deficiency of decay-accelerating factor accelerates murine T cell-mediated cardiac allograft rejection.

Myocardial ischemia and reperfusion injury is dependent on both IgM and mannose-binding lectin.

Mannose-binding lectin and its associated proteases (MASPs) mediate coagulation and its deficiency is a risk factor in developing complications from infection, including disseminated intravascular coagulation.

Complement 3 is involved with ventilator-induced lung injury.

Absence of mannose-binding lectin prevents hyperglycemic cardiovascular complications.

Mannose-binding lectin-associated serine protease-1 is a significant contributor to coagulation in a murine model of occlusive thrombosis.

Murine hyperglycemic vasculopathy and cardiomyopathy: whole-genome gene expression analysis predicts cellular targets and regulatory networks influenced by mannose binding lectin.

Targeting mannose-binding lectin confers long-lasting protection with a surprisingly wide therapeutic window in cerebral ischemia.

Endogenous and natural complement inhibitor attenuates myocardial injury and arterial thrombogenesis.

Human mannose-binding lectin inhibitor prevents myocardial injury and arterial thrombogenesis in a novel animal model.

Human mannose-binding lectin inhibitor prevents Shiga toxin-induced renal injury.