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Department of Physiology and Cell Biology,
Faculty of Health Sciences,
The Zlotowski Center for Neuroscience,
Department of Physiology and Cell Biology, Faculty of Health Sciences
Adrian Israelson has not added Biography.
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A chemical chaperone-based drug candidate is effective in a mouse model of amyotrophic lateral sclerosis (ALS).
ChemMedChem May, 2015 | Pubmed ID: 25772747
In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death.
The Biochemical journal Jan, 2004 | Pubmed ID: 14561215
Oligomeric states of the voltage-dependent anion channel and cytochrome c release from mitochondria.
The Biochemical journal Feb, 2005 | Pubmed ID: 15456403
Fluoxetine (Prozac) interaction with the mitochondrial voltage-dependent anion channel and protection against apoptotic cell death.
FEBS letters Sep, 2005 | Pubmed ID: 16139271
A photoactivable probe for calcium binding proteins.
Chemistry & biology Nov, 2005 | Pubmed ID: 16298296
Localization of the voltage-dependent anion channel-1 Ca2+-binding sites.
Cell calcium Mar, 2007 | Pubmed ID: 16930689
Azido ruthenium: a new photoreactive probe for calcium-binding proteins.
Nature protocols , 2006 | Pubmed ID: 17406221
Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1.
Cell calcium Feb, 2008 | Pubmed ID: 17590433
Hexokinase-I protection against apoptotic cell death is mediated via interaction with the voltage-dependent anion channel-1: mapping the site of binding.
The Journal of biological chemistry May, 2008 | Pubmed ID: 18308720
The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins.
Journal of cell science Jun, 2009 | Pubmed ID: 19461077
Misfolded mutant SOD1 directly inhibits VDAC1 conductance in a mouse model of inherited ALS.
Neuron Aug, 2010 | Pubmed ID: 20797535
ALS-linked mutant superoxide dismutase 1 (SOD1) alters mitochondrial protein composition and decreases protein import.
Proceedings of the National Academy of Sciences of the United States of America Dec, 2010 | Pubmed ID: 21078990
New fluorescent reagents specific for Ca(2+)-binding proteins.
Biochemical and biophysical research communications Sep, 2012 | Pubmed ID: 22925895
Why lithium studies for ALS treatment should not be halted prematurely.
Frontiers in neuroscience , 2014 | Pubmed ID: 25228854
Macrophage migration inhibitory factor as a chaperone inhibiting accumulation of misfolded SOD1.
Neuron Apr, 2015 | Pubmed ID: 25801706
Macrophage migration inhibitory factor as a component of selective vulnerability of motor neurons in ALS.
Rare diseases (Austin, Tex.) , 2015 | Pubmed ID: 26459694
Superoxide Dismutase 1 (SOD1)-Derived Peptide Inhibits Amyloid Aggregation of Familial Amyotrophic Lateral Sclerosis SOD1 Mutants.
ACS chemical neuroscience Nov, 2016 | Pubmed ID: 27540759
Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALS.
Proceedings of the National Academy of Sciences of the United States of America Sep, 2016 | Pubmed ID: 27551074
Ben-Gurion University of the Negev
Tom Shani1,
Moshe Levy1,
Adrian Israelson1,2
1Department of Physiology and Cell Biology, Faculty of Health Sciences, Ben-Gurion University of the Negev,
2The Zlotowski Center for Neuroscience, Ben-Gurion University of the Negev
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