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Division of Biomedical and Life Sciences
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Involvement of heat shock proteins on Mn-induced toxicity in Caenorhabditis elegans.
BMC pharmacology & toxicology 11, 2016 | Pubmed ID: 27802836
The SKN-1/Nrf2 transcription factor can protect against oxidative stress and increase lifespan in C. elegans by distinct mechanisms.
Aging cell 10, 2017 | Pubmed ID: 28612944
Coupling of Rigor Mortis and Intestinal Necrosis during C. elegans Organismal Death.
Cell reports 03, 2018 | Pubmed ID: 29514100
A parthenogenetic quasi-program causes teratoma-like tumors during aging in wild-type .
NPJ aging and mechanisms of disease , 2018 | Pubmed ID: 29928508
C. elegans Eats Its Own Intestine to Make Yolk Leading to Multiple Senescent Pathologies.
Current biology : CB 08, 2018 | Pubmed ID: 30100339
Autophagy promotes visceral aging in wild-type C. elegans.
Autophagy 04, 2019 | Pubmed ID: 30700231
New label-free automated survival assays reveal unexpected stress resistance patterns during C. elegans aging.
Aging cell 10, 2019 | Pubmed ID: 31309734
Gene therapy-mediated enhancement of protective protein expression for the treatment of Alzheimer's disease.
Brain research 02, 2021 | Pubmed ID: 33422539
BACE1 Overexpression Reduces SH-SY5Y Cell Viability Through a Mechanism Distinct from Amyloid-β Peptide Accumulation: Beta Prime-Mediated Competitive Depletion of sAβPPα.
Journal of Alzheimer's disease : JAD , 2022 | Pubmed ID: 35180123
Lancaster University
Irtiqa Ali*,1,
Jack Martin*,1,
Alejandra Zárate-Potes1,
Alexandre Benedetto1
1Division of Biomedical and Life Sciences, Lancaster University
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