Victoria Auerbuch

Impact of host membrane pore formation by the Yersinia pseudotuberculosis type III secretion system on the macrophage innate immune response.

An NF-κB-based high-throughput screen identifies piericidins as inhibitors of the Yersinia pseudotuberculosis type III secretion system.

IscR is essential for yersinia pseudotuberculosis type III secretion and virulence.

Yersinia pseudotuberculosis YopD mutants that genetically separate effector protein translocation from host membrane disruption.

Bacterial iron-sulfur cluster sensors in mammalian pathogens.

Yersinia Type III Secretion System Master Regulator LcrF.

Erratum for Duncan et al., An NF-κB-Based High-Throughput Screen Identifies Piericidins as Inhibitors of the Yersinia pseudotuberculosis Type III Secretion System.

Hereditary Hemochromatosis Predisposes Mice to Yersinia pseudotuberculosis Infection Even in the Absence of the Type III Secretion System.

The Type III Secretion System Cleans up Its Act(in).

Bacterial internalization is required to trigger NIK-dependent NF-κB activation in response to the bacterial type three secretion system.

Piericidin A1 Blocks Ysc Type III Secretion System Needle Assembly.

Synthetic Cyclic Peptomers as Type III Secretion System Inhibitors.

Mouse Models of Yersiniosis.

Iron availability and oxygen tension regulate the Yersinia Ysc type III secretion system to enable disseminated infection.

The Type III Secretion System as a Tool for Studying Cytosolic Innate Immune Surveillance.

Developing Cyclic Peptomers as Broad-Spectrum Type III Secretion System Inhibitors in Gram-Negative Bacteria.