Weili Cai

Ectopic histone H3S10 phosphorylation causes chromatin structure remodeling in Drosophila.

RNA polymerase II-mediated transcription at active loci does not require histone H3S10 phosphorylation in Drosophila.

The COOH-terminal domain of the JIL-1 histone H3S10 kinase interacts with histone H3 and is required for correct targeting to chromatin.

Polytene chromosome squash methods for studying transcription and epigenetic chromatin modification in Drosophila using antibodies.

Chromator is required for proper microtubule spindle formation and mitosis in Drosophila.

Asator, a tau-tubulin kinase homolog in Drosophila localizes to the mitotic spindle.

Phosphorylation of SU(VAR)3-9 by the chromosomal kinase JIL-1.

JIL-1 and Su(var)3-7 interact genetically and counteract each other's effect on position-effect variegation in Drosophila.

Synthesis of Tb(OH)(3) nanowire arrays via a facile template-assisted hydrothermal approach.

The chromodomain-containing NH(2)-terminus of Chromator interacts with histone H1 and is required for correct targeting to chromatin.

The epigenetic H3S10 phosphorylation mark is required for counteracting heterochromatic spreading and gene silencing in Drosophila melanogaster.