Renate Renkawitz-Pohl

The formation of syncytia within the visceral musculature of the Drosophila midgut is dependent on duf, sns and mbc.

A new translational repression element and unusual transcriptional control regulate expression of don juan during Drosophila spermatogenesis.

Myoblast determination in the somatic and visceral mesoderm depends on Notch signalling as well as on milliways(mili(Alk)) as receptor for Jeb signalling.

kette and blown fuse interact genetically during the second fusion step of myogenesis in Drosophila.

CTCF is conserved from Drosophila to humans and confers enhancer blocking of the Fab-8 insulator.

Replacement by Drosophila melanogaster protamines and Mst77F of histones during chromatin condensation in late spermatids and role of sesame in the removal of these proteins from the male pronucleus.

In Drosophila melanogaster, the rolling pebbles isoform 6 (Rols6) is essential for proper Malpighian tubule morphology.

In Drosophila, don juan and don juan like encode proteins of the spermatid nucleus and the flagellum and both are regulated at the transcriptional level by the TAF II80 cannonball while translational repression is achieved by distinct elements.

Drosophila rolling pebbles colocalises and putatively interacts with alpha-Actinin and the Sls isoform Zormin in the Z-discs of the sarcomere and with Dumbfounded/Kirre, alpha-Actinin and Zormin in the terminal Z-discs.

Myoblast fusion in Drosophila melanogaster is mediated through a fusion-restricted myogenic-adhesive structure (FuRMAS).

Blown fuse regulates stretching and outgrowth but not myoblast fusion of the circular visceral muscles in Drosophila.

The Wiskott-Aldrich syndrome protein (WASP) is essential for myoblast fusion in Drosophila.

Transition from a nucleosome-based to a protamine-based chromatin configuration during spermiogenesis in Drosophila.

The Drosophila insulator proteins CTCF and CP190 link enhancer blocking to body patterning.

WASP and SCAR have distinct roles in activating the Arp2/3 complex during myoblast fusion.

Dumpy-30 family members as determinants of male fertility and interaction partners of metal-responsive transcription factor 1 (MTF-1) in Drosophila.

FuRMAS: triggering myoblast fusion in Drosophila.

Distinct functions of Mst77F and protamines in nuclear shaping and chromatin condensation during Drosophila spermiogenesis.

Histone H4 acetylation is essential to proceed from a histone- to a protamine-based chromatin structure in spermatid nuclei of Drosophila melanogaster.

Stress-induced PARP activation mediates recruitment of Drosophila Mi-2 to promote heat shock gene expression.

Drosophila Swiprosin-1/EFHD2 accumulates at the prefusion complex stage during Drosophila myoblast fusion.

The syncytial visceral and somatic musculature develops independently of β3-Tubulin during Drosophila embryogenesis, while maternally supplied β1-Tubulin is stable until the early steps of myoblast fusion.

Multinucleated smooth muscles and mononucleated as well as multinucleated striated muscles develop during establishment of the male reproductive organs of Drosophila melanogaster.

Transcriptional regulation of Profilin during wound closure in Drosophila larvae.

The Hydra FGFR, Kringelchen, partially replaces the Drosophila Heartless FGFR.

The bromodomain-containing protein tBRD-1 is specifically expressed in spermatocytes and is essential for male fertility.

Myosin heavy chain-like localizes at cell contact sites during Drosophila myoblast fusion and interacts in vitro with Rolling pebbles 7.

Three levels of regulation lead to protamine and Mst77F expression in Drosophila.

Subunits of the histone chaperone CAF1 also mediate assembly of protamine-based chromatin.

Chromatin dynamics during spermiogenesis.

H3K79 methylation: a new conserved mark that accompanies H4 hyperacetylation prior to histone-to-protamine transition in Drosophila and rat.

Tethering Membrane Fusion: Common and Different Players in Myoblasts and at the Synapse.

Distinct genetic programs guide Drosophila circular and longitudinal visceral myoblast fusion.