Aaron B. Baker

Vascular neointimal formation and signaling pathway activation in response to stent injury in insulin-resistant and diabetic animals.

Endothelial cells provide feedback control for vascular remodeling through a mechanosensitive autocrine TGF-beta signaling pathway.

Attenuation of inflammation and expansive remodeling by Valsartan alone or in combination with Simvastatin in high-risk coronary atherosclerotic plaques.

Heparanase alters arterial structure, mechanics, and repair following endovascular stenting in mice.

Pheochromocytoma-induced cardiomyopathy is modulated by the synergistic effects of cell-secreted factors.

The role of low endothelial shear stress in the conversion of atherosclerotic lesions from stable to unstable plaque.

Natural history of experimental coronary atherosclerosis and vascular remodeling in relation to endothelial shear stress: a serial, in vivo intravascular ultrasound study.

Augmentation of postswelling surgical sealant potential of adhesive hydrogels.

Aldehyde-amine chemistry enables modulated biosealants with tissue-specific adhesion.

Regulation of heparanase expression in coronary artery disease in diabetic, hyperlipidemic swine.

Stromal endothelial cells directly influence cancer progression.

Augmented expression and activity of extracellular matrix-degrading enzymes in regions of low endothelial shear stress colocalize with coronary atheromata with thin fibrous caps in pigs.

Syndecan-4 proteoliposomes enhance fibroblast growth factor-2 (FGF-2)-induced proliferation, migration, and neovascularization of ischemic muscle.

Heparanase regulates thrombosis in vascular injury and stent-induced flow disturbance.

The role of scaffold microarchitecture in engineering endothelial cell immunomodulation.

A multichannel dampened flow system for studies on shear stress-mediated mechanotransduction.

Analysis of a high-throughput cone-and-plate apparatus for the application of defined spatiotemporal flow to cultured cells.

Thin-capped atheromata with reduced collagen content in pigs develop in coronary arterial regions exposed to persistently low endothelial shear stress.

A novel system for studying mechanical strain waveform-dependent responses in vascular smooth muscle cells.

Overcoming disease-induced growth factor resistance in therapeutic angiogenesis using recombinant co-receptors delivered by a liposomal system.

Synergistic effect of local endothelial shear stress and systemic hypercholesterolemia on coronary atherosclerotic plaque progression and composition in pigs.

Loss of syndecan-1 induces a pro-inflammatory phenotype in endothelial cells with a dysregulated response to atheroprotective flow.

Syndecan-1 regulates vascular smooth muscle cell phenotype.

Synergistic effects of matrix nanotopography and stiffness on vascular smooth muscle cell function.