Alayna E. Loiselle

Remodeling of murine intrasynovial tendon adhesions following injury: MMP and neotendon gene expression.

TAK1 regulates cartilage and joint development via the MAPK and BMP signaling pathways.

Impact of Smad3 loss of function on scarring and adhesion formation during tendon healing.

Optimizing the osteogenic potential of adult stem cells for skeletal regeneration.

Osteoblast and osteocyte-specific loss of Connexin43 results in delayed bone formation and healing during murine fracture healing.

Bone marrow-derived matrix metalloproteinase-9 is associated with fibrous adhesion formation after murine flexor tendon injury.

Gap junction and hemichannel functions in osteocytes.

Specific biomimetic hydroxyapatite nanotopographies enhance osteoblastic differentiation and bone graft osteointegration.

Biophysical regulation of stem cell differentiation.

Connexin 43 deficiency desensitizes bone to the effects of mechanical unloading through modulation of both arms of bone remodeling.

Inhibition of GSK-3β rescues the impairments in bone formation and mechanical properties associated with fracture healing in osteoblast selective connexin 43 deficient mice.

Shifting paradigms on the role of connexin43 in the skeletal response to mechanical load.

Evidence for the role of connexin 43-mediated intercellular communication in the process of intracortical bone resorption via osteocytic osteolysis.

Integrative transcriptomic and proteomic analysis of osteocytic cells exposed to fluid flow reveals novel mechano-sensitive signaling pathways.

Parathyroid hormone 1-34 enhances extracellular matrix deposition and organization during flexor tendon repair.

Loss of the PGE2 receptor EP1 enhances bone acquisition, which protects against age and ovariectomy-induced impairments in bone strength.

Development of antisense oligonucleotide (ASO) technology against Tgf-β signaling to prevent scarring during flexor tendon repair.

Systemic EP4 Inhibition Increases Adhesion Formation in a Murine Model of Flexor Tendon Repair.

Biological Augmentation of Flexor Tendon Repair: A Challenging Cellular Landscape.

Low-Dose and Short-Duration Matrix Metalloproteinase 9 Inhibition Does Not Affect Adhesion Formation during Murine Flexor Tendon Healing.