Yuntao Wu

Gene transcription in HIV infection.

Early transcription from nonintegrated DNA in human immunodeficiency virus infection.

HIV-1 gene expression: lessons from provirus and non-integrated DNA.

No single homologous repeat region is essential for DNA replication of the baculovirus Autographa californica multiple nucleopolyhedrovirus.

Rev-dependent lentiviral expression vector.

Rev-dependent indicator T cell line.

Endogenous TGF-beta activation by reactive oxygen species is key to Foxp3 induction in TCR-stimulated and HIV-1-infected human CD4+CD25- T cells.

Human macrophages support persistent transcription from unintegrated HIV-1 DNA.

Selective killing of HIV-1-positive macrophages and T cells by the Rev-dependent lentivirus carrying anthrolysin O from Bacillus anthracis.

The second chance story of HIV-1 DNA: Unintegrated? Not a problem!

HIV envelope-CXCR4 signaling activates cofilin to overcome cortical actin restriction in resting CD4 T cells.

Cofilin activation in peripheral CD4 T cells of HIV-1 infected patients: a pilot study.

The co-receptor signaling model of HIV-1 pathogenesis in peripheral CD4 T cells.

Measurement of human immunodeficiency virus type 1 preintegration transcription by using Rev-dependent Rev-CEM cells reveals a sizable transcribing DNA population comparable to that from proviral templates.

Chemokine receptor signaling and HIV infection.

The HIV envelope but not VSV glycoprotein is capable of mediating HIV latent infection of resting CD4 T cells.

Chemokine coreceptor signaling in HIV-1 infection and pathogenesis.

Chemokine control of HIV-1 infection: beyond a binding competition.

Control of the differentiation of regulatory T cells and T(H)17 cells by the DNA-binding inhibitor Id3.

Effects of microtubule modulators on HIV-1 infection of transformed and resting CD4 T cells.

LIM kinase 1 modulates cortical actin and CXCR4 cycling and is activated by HIV-1 to initiate viral infection.

Spinoculation triggers dynamic actin and cofilin activity that facilitates HIV-1 infection of transformed and resting CD4 T cells.

Study on the luminescence and energy level of lanthanide ions in Lu(0.8)Sc(0.2)BO3 host.

Development of a nonintegrating Rev-dependent lentiviral vector carrying diphtheria toxin A chain and human TRAF6 to target HIV reservoirs.

The trinity of the cortical actin in the initiation of HIV-1 infection.

A dichotomy in cortical actin and chemotactic actin activity between human memory and naive T cells contributes to their differential susceptibility to HIV-1 infection.

HIV gp120 is an aberrant chemoattractant for blood resting CD4 T cells.

Involvement of LIM kinase 1 in actin polarization in human CD4 T cells.

The BET bromodomain inhibitor JQ1 activates HIV latency through antagonizing Brd4 inhibition of Tat-transactivation.

Inhibition of HIV replication in vitro by clinical immunosuppressants and chemotherapeutic agents.

Genistein interferes with SDF-1- and HIV-mediated actin dynamics and inhibits HIV infection of resting CD4 T cells.

Novel anti-HIV therapeutics targeting chemokine receptors and actin regulatory pathways.

HIV-1 triggers WAVE2 phosphorylation in primary CD4 T cells and macrophages, mediating Arp2/3-dependent nuclear migration.

Viral exploitation of actin: force-generation and scaffolding functions in viral infection.

Chemokine receptor CCR5 antagonist maraviroc: medicinal chemistry and clinical applications.

Bis-aryl urea derivatives as potent and selective LIM kinase (Limk) inhibitors.

BRCA1 functions as a novel transcriptional cofactor in HIV-1 infection.

Therapeutic doses of irradiation activate viral transcription and induce apoptosis in HIV-1 infected cells.

Real-Time Imaging of Single HIV-1 Disassembly with Multicolor Viral Particles.

Flow Cytometry-Based Quantification of HIV-Induced T Cell Chemotactic Response.

Single-Particle Tracking of Human Immunodeficiency Virus Type 1 Productive Entry into Human Primary Macrophages.

Discovery of Novel Small-Molecule Inhibitors of LIM Domain Kinase for Inhibiting HIV-1.

Elevated interferon-γ-induced protein 10 and its receptor CXCR3 impair NK cell function during HIV infection.

Persistence of SIV in the brain of SIV-infected Chinese rhesus macaques with or without antiretroviral therapy.

AID recruits the RNA exosome to degrade HIV-1 nascent transcripts through interaction with the Tat-P-TEFb-TAR RNP complex.

Tat controls transcriptional persistence of unintegrated HIV genome in primary human macrophages.

Cofilin hyperactivation in HIV infection and targeting the cofilin pathway using an anti-αβ integrin antibody.

Proteomic profiling of HIV-1 infection of human CD4 T cells identifies PSGL-1 as an HIV restriction factor.

Relationships Between HIV-Mediated Chemokine Coreceptor Signaling, Cofilin Hyperactivation, Viral Tropism Switch and HIV-Mediated CD4 Depletion.

Real-time imaging of individual virion-triggered cortical actin dynamics for human immunodeficiency virus entry into resting CD4 T cells.

Compensation of ACE2 Function for Possible Clinical Management of 2019-nCoV-Induced Acute Lung Injury.

Understanding SARS-CoV-2-Mediated Inflammatory Responses: From Mechanisms to Potential Therapeutic Tools.

SARS-CoV-2 is an appropriate name for the new coronavirus.

PSGL-1 restricts HIV-1 infectivity by blocking virus particle attachment to target cells.

Conditionally Reprogrammed Human Normal Airway Epithelial Cells at ALI: A Physiological Model for Emerging Viruses.

Targeted delivery of lopinavir to HIV reservoirs in the mesenteric lymphatic system by lipophilic ester prodrug approach.

Alterations of the gut bacterial microbiota in rhesus macaques with SIV infection and on short- or long-term antiretroviral therapy.

PSGL-1 Inhibits the Incorporation of SARS-CoV and SARS-CoV-2 Spike Glycoproteins into Pseudovirions and Impairs Pseudovirus Attachment and Infectivity.

A traditional medicine, respiratory detox shot (RDS), inhibits the infection of SARS-CoV, SARS-CoV-2, and the influenza A virus in vitro.

Identification of the SHREK Family of Proteins as Broad-Spectrum Host Antiviral Factors.

IP-10 Promotes Latent HIV Infection in Resting Memory CD4 T Cells LIMK-Cofilin Pathway.

Prestimulation of CD2 confers resistance to HIV-1 latent infection in blood resting CD4 T cells.

Exosomes originating from infection with the cytoplasmic single-stranded RNA virus Rift Valley fever virus (RVFV) protect recipient cells by inducing RIG-I mediated IFN-B response that leads to activation of autophagy.

Development of a hybrid alphavirus-SARS-CoV-2 pseudovirion for rapid quantification of neutralization antibodies and antiviral drugs.

Suppressing fatty acid synthase by type I interferon and chemical inhibitors as a broad spectrum anti-viral strategy against SARS-CoV-2.

HIV Preintegration Transcription and Host Antagonism.