Vijayalakshmi Santhakumar

Mossy cells in epilepsy: rigor mortis or vigor mortis?

Postsynaptic effects of GABAergic synaptic diversity: regulation of neuronal excitability by changes in IPSC variance.

H-channels in epilepsy: new targets for seizure control?

Post-traumatic hyperexcitability is not caused by impaired buffering of extracellular potassium.

Rapid deletion of mossy cells does not result in a hyperexcitable dentate gyrus: implications for epileptogenesis.

Impact of heterogeneous perisomatic IPSC populations on pyramidal cell firing rates.

Plasticity of interneuronal species diversity and parameter variance in neurological diseases.

Role of mossy fiber sprouting and mossy cell loss in hyperexcitability: a network model of the dentate gyrus incorporating cell types and axonal topography.

Contributions of the GABAA receptor alpha6 subunit to phasic and tonic inhibition revealed by a naturally occurring polymorphism in the alpha6 gene.

Topological determinants of epileptogenesis in large-scale structural and functional models of the dentate gyrus derived from experimental data.

Ethanol acts directly on extrasynaptic subtypes of GABAA receptors to increase tonic inhibition.

Modeling the dentate gyrus.

Decrease in tonic inhibition contributes to increase in dentate semilunar granule cell excitability after brain injury.

Status epilepticus enhances tonic GABA currents and depolarizes GABA reversal potential in dentate fast-spiking basket cells.

A reinforcing circuit action of extrasynaptic GABAA receptor modulators on cerebellar granule cell inhibition.

Seizure-induced alterations in fast-spiking basket cell GABA currents modulate frequency and coherence of gamma oscillation in network simulations.

Distinct effect of impact rise times on immediate and early neuropathology after brain injury in juvenile rats.

Electrophysiological monitoring of injury progression in the rat cerebellar cortex.

Long-lasting suppression of acoustic startle response after mild traumatic brain injury.

Toll-like receptor 4 enhancement of non-NMDA synaptic currents increases dentate excitability after brain injury.

Dentate total molecular layer interneurons mediate cannabinoid-sensitive inhibition.

Fluid percussion injury device for the precise control of injury parameters.

Functional Reduction in Cannabinoid-Sensitive Heterotypic Inhibition of Dentate Basket Cells in Epilepsy: Impact on Network Rhythms.

Dentate cannabinoid-sensitive interneurons undergo unique and selective strengthening of mutual synaptic inhibition in experimental epilepsy.

Facilitating Mitochondrial Calcium Uptake Improves Activation-Induced Cerebral Blood Flow and Behavior after mTBI.

High Ca(2+) Influx During Traumatic Brain Injury Leads to Caspase-1-Dependent Neuroinflammation and Cell Death.

Traumatic brain injury induced matrix metalloproteinase2 cleaves CXCL12α (stromal cell derived factor 1α) and causes neurodegeneration.