Lorena Benedetti

SPARC endogenous level, rather than fibroblast-produced SPARC or stroma reorganization induced by SPARC, is responsible for melanoma cell growth.

The role of the matricellular protein SPARC in the dynamic interaction between the tumor and the host.

The role of the matricellular protein SPARC in the dynamic interaction between the tumor and the host.

Matricellular proteins and inflammatory cells: a task force to promote or defeat cancer?

SPARC downregulation attenuates the profibrogenic response of hepatic stellate cells induced by TGF-β1 and PDGF.

SPARC promotes cathepsin B-mediated melanoma invasiveness through a collagen I/α2β1 integrin axis.

A Tumor-stroma Targeted Oncolytic Adenovirus Replicated in Human Ovary Cancer Samples and Inhibited Growth of Disseminated Solid Tumors in Mice.

Subtype-specific overexpression of the Rac-GEF P-REX1 in breast cancer is associated with promoter hypomethylation.

ICln: a new regulator of non-erythroid 4.1R localisation and function.

Customized patterned substrates for highly versatile correlative light-scanning electron microscopy.

INaP selective inhibition reverts precocious inter- and motorneurons hyperexcitability in the Sod1-G93R zebrafish ALS model.

Patients with genetically heterogeneous synchronous colorectal cancer carry rare damaging germline mutations in immune-related genes.

MEGA-V: detection of variant gene sets in patient cohorts.

Predictive Outcomes for HER2-enriched Cancer Using Growth and Metastasis Signatures Driven By SPARC.

Epilepsy and intellectual disability linked protein Shrm4 interaction with GABABRs shapes inhibitory neurotransmission.

Synthetic lethal interaction between the tumour suppressor STAG2 and its paralog STAG1.