Wayne S. Sossin

Phosphoinositide-dependent kinase phosphorylation of protein kinase C Apl II increases during intermediate facilitation in aplysia.

Oxidation induces autonomous activation of protein kinase C Apl I, but not protein kinase C Apl II in homogenates of Aplysia neurons.

Protein kinase C isoforms are translocated to microtubules in neurons.

An activity-dependent switch to cap-independent translation triggered by eIF4E dephosphorylation.

In vitro autophosphorylation of protein kinase C isozymes.

Phosphopeptide-specific antibodies to protein kinase C.

Differential regulation of transmitter release by alternatively spliced forms of synaptotagmin I.

依赖于活动的调节神经激素合成及对海蜗牛的生殖行为的影响。

Identification and characterization of a novel C2B splice variant of synaptotagmin I.

Characterization of a novel molluskan tyrosine kinase receptor that inhibits neurite regeneration.

5-HT stimulates eEF2 dephosphorylation in a rapamycin-sensitive manner in Aplysia neurites.

ApTrkl, a Trk-like receptor, mediates serotonin- dependent ERK activation and long-term facilitation in Aplysia sensory neurons.

Ltrk is differentially expressed in developing and adult neurons of the Lymnaea central nervous system.

Characterization of an RNA granule from developing brain.

Mnk is a negative regulator of cap-dependent translation in Aplysia neurons.

Tracing the evolution and function of the Trk superfamily of receptor tyrosine kinases.

Isoform specificity of PKC translocation in living Aplysia sensory neurons and a role for Ca2+-dependent PKC APL I in the induction of intermediate-term facilitation.

PKC modulation of transmitter release by SNAP-25 at sensory-to-motor synapses in aplysia.

Serotonin increases phosphorylation of synaptic 4EBP through TOR, but eukaryotic initiation factor 4E levels do not limit somatic cap-dependent translation in aplysia neurons.

Intracellular trafficking of RNA in neurons.

Isoform specificity of protein kinase Cs in synaptic plasticity.

Something old, something new: BDNF-induced neuron survival requires TRPC channel function.

Defining memories by their distinct molecular traces.

The Essence of Memory. Preface.

Molecular memory traces.

Forskolin induction of late-LTP and up-regulation of 5' TOP mRNAs translation via mTOR, ERK, and PI3K in hippocampal pyramidal cells.

Physiological role for phosphatidic acid in the translocation of the novel protein kinase C Apl II in Aplysia neurons.

Mechanisms regulating ApTrkl, a Trk-like receptor in Aplysia sensory neurons.

Translational control of long-lasting synaptic plasticity and memory.

The atypical protein kinase C in Aplysia can form a protein kinase M by cleavage.

Combinations of DEAD box proteins distinguish distinct types of RNA: protein complexes in neurons.

Role of protein kinase C in the induction and maintenance of serotonin-dependent enhancement of the glutamate response in isolated siphon motor neurons of Aplysia californica.

Synapse- and stimulus-specific local translation during long-term neuronal plasticity.

PKC differentially translocates during spaced and massed training in Aplysia.

Evolutionary conservation of the signaling proteins upstream of cyclic AMP-dependent kinase and protein kinase C in gastropod mollusks.

Ribosomal protein S6 kinase is a critical downstream effector of the target of rapamycin complex 1 for long-term facilitation in Aplysia.

Postnatal deamidation of 4E-BP2 in brain enhances its association with raptor and alters kinetics of excitatory synaptic transmission.

Mechanisms of translational regulation in synaptic plasticity.

Aplysia cell adhesion molecule and a novel protein kinase C activity in the postsynaptic neuron are required for presynaptic growth and initial formation of specific synapses.

Regulation of protein kinase C Apl II by serotonin receptors in Aplysia.

The significance of organellar proteomics for the nervous system.

Translation of 5' terminal oligopyrimidine tract (5'TOP) mRNAs in Aplysia Californica is regulated by the target of rapamycin (TOR).

Compartment-specific, differential regulation of eukaryotic elongation factor 2 and its kinase within Aplysia sensory neurons.

Staufen 2 regulates mGluR long-term depression and Map1b mRNA distribution in hippocampal neurons.

A new mechanism of action of a C2 domain-derived novel PKC inhibitor peptide.

Inhibitory responses in Aplysia pleural sensory neurons act to block excitability, transmitter release, and PKC Apl II activation.

The rates of protein synthesis and degradation account for the differential response of neurons to spaced and massed training protocols.