Laszlo Perlaky

Regression of experimental medulloblastoma following transfer of HER2-specific T cells.

Overexpressed TP73 induces apoptosis in medulloblastoma.

Direct orthotopic transplantation of fresh surgical specimen preserves CD133+ tumor cells in clinically relevant mouse models of medulloblastoma and glioma.

Expression, cellular localization, and enzymatic activities of RNA helicase II/Gu(beta).

Drug-induced translocation of nucleolar proteins fused to green fluorescent protein.

Frequent amplification and rearrangement of chromosomal bands 6p12-p21 and 17p11.2 in osteosarcoma.

Phenylbutyrate and phenylacetate induce differentiation and inhibit proliferation of human medulloblastoma cells.

Comparison of ethanol versus formalin fixation on preservation of histology and RNA in laser capture microdissected brain tissues.

Genome-wide array comparative genomic hybridization analysis reveals distinct amplifications in osteosarcoma.

Expression analysis of juvenile pilocytic astrocytomas by oligonucleotide microarray reveals two potential subgroups.

Expression profiles of osteosarcoma that can predict response to chemotherapy.

Valproic acid induces growth arrest, apoptosis, and senescence in medulloblastomas by increasing histone hyperacetylation and regulating expression of p21Cip1, CDK4, and CMYC.

Optimising the use of TRIzol-extracted proteins in surface enhanced laser desorption/ ionization (SELDI) analysis.

Identification of a plasma proteomic signature to distinguish pediatric osteosarcoma from benign osteochondroma.

Medulloblastoma outcome is adversely associated with overexpression of EEF1D, RPL30, and RPS20 on the long arm of chromosome 8.

Genome-wide allelic imbalance analysis of pediatric gliomas by single nucleotide polymorphic allele array.

Cell cycle regulator gene CDC5L, a potential target for 6p12-p21 amplicon in osteosarcoma.

Immunotherapy for osteosarcoma: genetic modification of T cells overcomes low levels of tumor antigen expression.

Nucleolar changes and fibrillarin redistribution following apatone treatment of human bladder carcinoma cells.

Crosstalk between medulloblastoma cells and endothelium triggers a strong chemotactic signal recruiting T lymphocytes to the tumor microenvironment.