Department of Cell and Molecular Biology
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Activation of hypoxia-inducible factor 1alpha: posttranscriptional regulation and conformational change by recruitment of the Arnt transcription factor.
Proceedings of the National Academy of Sciences of the United States of America May, 1997 | Pubmed ID: 9159130
Hypoxia requires notch signaling to maintain the undifferentiated cell state.
Developmental cell Nov, 2005 | Pubmed ID: 16256737
Interaction with factor inhibiting HIF-1 defines an additional mode of cross-coupling between the Notch and hypoxia signaling pathways.
Proceedings of the National Academy of Sciences of the United States of America Mar, 2008 | Pubmed ID: 18299578
Specific ligation to double-stranded RNA for analysis of cellular RNA::RNA interactions.
Nucleic acids research Sep, 2008 | Pubmed ID: 18628292
Complex regulation of the transactivation function of hypoxia-inducible factor-1 alpha by direct interaction with two distinct domains of the CREB-binding protein/p300.
The Journal of biological chemistry Jan, 2010 | Pubmed ID: 19880525
Regulation of CDKN2B expression by interaction of Arnt with Miz-1--a basis for functional integration between the HIF and Myc gene regulatory pathways.
Molecular cancer Mar, 2014 | Pubmed ID: 24618291
Regulation of hypoxia-inducible gene expression after HIF activation.
Experimental cell research 07, 2017 | Pubmed ID: 28286304
A Molecular Mechanism To Switch the Aryl Hydrocarbon Receptor from a Transcription Factor to an E3 Ubiquitin Ligase.
Molecular and cellular biology Jul, 2017 | Pubmed ID: 28416634
A Notch-independent mechanism contributes to the induction of Hes1 gene expression in response to hypoxia in P19 cells.
Experimental cell research 09, 2017 | Pubmed ID: 28602625
Interaction between von Hippel-Lindau Protein and Fatty Acid Synthase Modulates Hypoxia Target Gene Expression.
Scientific reports Aug, 2017 | Pubmed ID: 28775317
Author Correction: Interaction between von Hippel-Lindau Protein and Fatty Acid Synthase Modulates Hypoxia Target Gene Expression.
Scientific reports Jul, 2018 | Pubmed ID: 30030449
Xiaofeng Zheng1,2,
Calvin Qing Wei Ho1,
Xiaowei Zheng3,
Kian Leong Lee4,
Katarina Gradin5,
Teresa S. Pereira3,
Per-Olof Berggren1,2,3,
Yusuf Ali1,2
1Lee Kong Chian School of Medicine, Nanyang Technological University,
2Singapore Eye Research Institute (SERI), Singapore General Hospital,
3The Rolf Luft Research Center for Diabetes and Endocrinology, Karolinska Institutet, Karolinska University Hospital,
4Cancer and Stem Cell Biology Program, Duke-NUS Medical School,
5Department of Cell and Molecular Biology, Karolinska Institutet
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