Dilson E. Rassier

Pre-power stroke cross bridges contribute to force during stretch of skeletal muscle myofibrils.

Staircase but not posttetanic potentiation in rat muscle after spinal cord hemisection.

The mechanical behavior of individual sarcomeres of myofibrils isolated from rabbit psoas muscle.

Molecular basis of force development by skeletal muscles during and after stretch.

Effects of blebbistatin and Ca2+ concentration on force produced during stretch of skeletal muscle fibers.

Striated muscles: from molecules to cells.

Contractile characteristics of sarcomeres arranged in series or mechanically isolated from myofibrils.

Stretch and shortening of skeletal muscles activated along the ascending limb of the force-length relation.

A technique for simultaneous measurement of force and overlap between single muscle filaments of myosin and actin.

Force produced by isolated sarcomeres and half-sarcomeres after an imposed stretch.

Pre-power-stroke cross-bridges contribute to force transients during imposed shortening in isolated muscle fibers.

The mechanisms of the residual force enhancement after stretch of skeletal muscle: non-uniformity in half-sarcomeres and stiffness of titin.

Arginylation regulates myofibrils to maintain heart function and prevent dilated cardiomyopathy.

Residual force enhancement in skeletal muscles: one sarcomere after the other.

Forces measured with micro-fabricated cantilevers during actomyosin interactions produced by filaments containing different myosin isoforms and loop 1 structures.

Forces measured with micro-fabricated cantilevers during actomyosin interactions produced by filaments containing different myosin isoforms and loop 1 structures.

The effects of Ca2+ and MgADP on force development during and after muscle length changes.

Force produced after stretch in sarcomeres and half-sarcomeres isolated from skeletal muscles.

Nitrosative modifications of the Ca2+ release complex and actin underlie arthritis-induced muscle weakness.

Arginylation of myosin heavy chain regulates skeletal muscle strength.

Non-crossbridge forces in activated striated muscles: a titin dependent mechanism of regulation?

Masticatory muscles of mouse do not undergo atrophy in space.

Thixotropy and rheopexy of muscle fibers probed using sinusoidal oscillations.

Residual force depression in single sarcomeres is abolished by MgADP-induced activation.

MgADP activation contributes to force enhancement during fast stretch of isolated skeletal myofibrils.

The increase in non-cross-bridge forces after stretch of activated striated muscle is related to titin isoforms.

Reduced passive force in skeletal muscles lacking protein arginylation.

Reduction in single muscle fiber rate of force development with aging is not attenuated in world class older masters athletes.

Two Kinases to Soften the Heart.

Reactive oxygen/nitrogen species and contractile function in skeletal muscle during fatigue and recovery.

Prolonged controlled mechanical ventilation in humans triggers myofibrillar contractile dysfunction and myofilament protein loss in the diaphragm.

Reply to "Letter to the editor: Titin-actin interaction: the report of its death was an exaggeration".

Posttranslational Arginylation Regulates Striated Muscle Function.

Length-dependent Ca2+ activation in skeletal muscle fibers from mammalians.

Reply to "Letter to the editor: Comments on Cornachione et al. (2016): "The increase in non-cross-bridge forces after stretch of activated striated muscle is related to titin isoforms".

Residual force enhancement is regulated by titin in skeletal and cardiac myofibrils.

Sarcomere mechanics in striated muscles: from molecules to sarcomeres to cells.

Prolonged force depression after mechanically demanding contractions is largely independent of Ca and reactive oxygen species.

Microfluidic perfusion shows intersarcomere dynamics within single skeletal muscle myofibrils.

Sarcomere Stiffness during Stretching and Shortening of Rigor Skeletal Myofibrils.

Dysfunctional sarcomere contractility contributes to muscle weakness in ACTA1-related nemaline myopathy (NEM3).

Do Actomyosin Single-Molecule Mechanics Data Predict Mechanics of Contracting Muscle?

Blebbistatin Effects Expose Hidden Secrets in the Force-Generating Cycle of Actin and Myosin.

Hypertrophic cardiomyopathy R403Q mutation in rabbit β-myosin reduces contractile function at the molecular and myofibrillar levels.

Cleavage of loops 1 and 2 in skeletal muscle heavy meromyosin (HMM) leads to a decreased function.

Nonlinear Actomyosin Elasticity in Muscle?

Protein arginylation of cytoskeletal proteins in the muscle: modifications modifying function.

The effects of fatigue and oxidation on contractile function of intact muscle fibers and myofibrils isolated from the mouse diaphragm.

Oxidative hotspots on actin promote skeletal muscle weakness in rheumatoid arthritis.

Mechanical ventilation causes diaphragm dysfunction in newborn lambs.

High-speed AFM reveals subsecond dynamics of cardiac thin filaments upon Ca activation and heavy meromyosin binding.

Force generated by myosin cross-bridges is reduced in myofibrils exposed to ROS/RNS.

Skeletal MyBP-C isoforms tune the molecular contractility of divergent skeletal muscle systems.

The load dependence and the force-velocity relation in intact myosin filaments from skeletal and smooth muscles.

KBTBD13 is an actin-binding protein that modulates muscle kinetics.

Extraction of Thick Filaments in Individual Sarcomeres Affects Force Production by Single Myofibrils.