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Institute for Regenerative Medicine
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Oxidative stress and altered mitochondrial protein expression in the absence of amyloid-β and tau pathology in iPSC-derived neurons from sporadic Alzheimer's disease patients.
Stem cell research 03, 2018 | Pubmed ID: 29414602
The secreted APP ectodomain sAPPα, but not sAPPβ, protects neurons against Aβ oligomer-induced dendritic spine loss and increased tau phosphorylation.
Molecular brain 03, 2019 | Pubmed ID: 30922360
Alzheimer's in a dish - induced pluripotent stem cell-based disease modeling.
Translational neurodegeneration , 2019 | Pubmed ID: 31338163
A Practical Guide to the Automated Analysis of Vascular Growth, Maturation and Injury in the Brain.
Frontiers in neuroscience , 2020 | Pubmed ID: 32265643
Familial Alzheimer's disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system.
PloS one , 2020 | Pubmed ID: 32966331
Characterization of the Blood Brain Barrier Disruption in the Photothrombotic Stroke Model.
Frontiers in physiology , 2020 | Pubmed ID: 33262704
Increased maturation of iPSC-derived neurons in a hydrogel-based 3D culture.
Journal of neuroscience methods 08, 2021 | Pubmed ID: 34126141
APOE2, E3, and E4 differentially modulate cellular homeostasis, cholesterol metabolism, and inflammatory response in isogenic iPSC-derived astrocytes.
Stem cell reports Jan, 2022 | Pubmed ID: 34919811
University of Zurich
University of Zurich and ETH Zurich
Rebecca Z. Weber1,2,
Chantal Bodenmann1,
Daniela Uhr1,
Kathrin J. Zürcher1,
Debora Wanner1,
Melanie Generali1,
Roger M. Nitsch1,
Ruslan Rust*,1,2,
Christian Tackenberg*,1,2
1Institute for Regenerative Medicine, University of Zurich,
2Neuroscience Center Zurich, University of Zurich and ETH Zurich
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