Pampee P. Young

VEGF increases engraftment of bone marrow-derived endothelial progenitor cells (EPCs) into vasculature of newborn murine recipients.

Biodistribution and efficacy of donor T lymphocytes in a murine model of lysosomal storage disease.

Glucocorticoid inhibition of SP-A gene expression in lung type II cells is mediated via the TTF-1-binding element.

Autoantibody formation after alloimmunization: are blood transfusions a risk factor for autoimmune hemolytic anemia?

VEGF disrupts the neonatal blood-brain barrier and increases life span after non-ablative BMT in a murine model of congenital neurodegeneration caused by a lysosomal enzyme deficiency.

Blood donor white blood cell reduction filters as a source of human peripheral blood-derived endothelial progenitor cells.

Enrichment of genes in the aortic intima that are associated with stratified epithelium: implications of underlying biomechanical and barrier properties of the arterial intima.

The origin and in vivo significance of murine and human culture-expanded endothelial progenitor cells.

VEGF and PlGF promote adult vasculogenesis by enhancing EPC recruitment and vessel formation at the site of tumor neovascularization.

Biologic properties of endothelial progenitor cells and their potential for cell therapy.

The MD/PhD pathway to a career in laboratory medicine.

Adenosine receptor-mediated adhesion of endothelial progenitors to cardiac microvascular endothelial cells.

Low-dose growth hormone administration mobilizes endothelial progenitor cells in healthy adults.

Damage control hematology: the impact of a trauma exsanguination protocol on survival and blood product utilization.

The effects of growth hormone and insulin-like growth factor-1 on the aging cardiovascular system and its progenitor cells.

Optimizing outcomes in damage control resuscitation: identifying blood product ratios associated with improved survival.

Regulation of the atheroma-enriched protein, SPRR3, in vascular smooth muscle cells through cyclic strain is dependent on integrin alpha1beta1/collagen interaction.

The Wnt modulator sFRP2 enhances mesenchymal stem cell engraftment, granulation tissue formation and myocardial repair.

Low levels of tumor necrosis factor alpha increase tumor growth by inducing an endothelial phenotype of monocytes recruited to the tumor site.

Predefined massive transfusion protocols are associated with a reduction in organ failure and postinjury complications.

Biomechanical stress induces novel arterial intima-enriched genes: implications for vascular adaptation to stress.

A novel model-gel-tissue assay analysis for comparing tumor elastic properties to collagen content.

Hyperkalemia following massive transfusion in trauma.

Room for (performance) improvement: provider-related factors associated with poor outcomes in massive transfusion.

Proteomic analysis of osteogenic sarcoma: association of tumour necrosis factor with poor prognosis.

Emergency department blood transfusion predicts early massive transfusion and early blood component requirement.

Creation, implementation, and maturation of a massive transfusion protocol for the exsanguinating trauma patient.

Atheromas feel the pressure: biomechanical stress and atherosclerosis.

Multicenter validation of a simplified score to predict massive transfusion in trauma.

sFRP2 suppression of bone morphogenic protein (BMP) and Wnt signaling mediates mesenchymal stem cell (MSC) self-renewal promoting engraftment and myocardial repair.

Pyrvinium, a potent small molecule Wnt inhibitor, promotes wound repair and post-MI cardiac remodeling.

Myeloid cells in cancer progression: unique subtypes and their roles in tumor growth, vascularity, and host immune suppression.

TNFalpha accelerates monocyte to endothelial transdifferentiation in tumors by the induction of integrin alpha5 expression and adhesion to fibronectin.

Massive transfusion protocols for patients with substantial hemorrhage.

A window of opportunity: the aggressive use of plasma in early resuscitation.

Mesenchymal stem cell transplantation for the infarcted heart: a role in minimizing abnormalities in cardiac-specific energy metabolism.

Transfusion medicine illustrated. Profound piperacillin-mediated drug-induced immune hemolysis in a patient with cystic fibrosis.

Lessons from Genetically Altered Mesenchymal Stem Cells (MSCs); Candidates for Improved MSC-Directed Myocardial Repair.

Molecular mediators of mesenchymal stem cell biology.

Pyrvinium, a potent small molecule Wnt inhibitor, increases engraftment and inhibits lineage commitment of mesenchymal stem cells (MSCs).