Chaojun Tang

Standardization of a well-controlled in vivo mouse model of thrombus formation induced by mechanical injury.

Vascular Semaphorin 7A Upregulation by Disturbed Flow Promotes Atherosclerosis Through Endothelial β1 Integrin.

Coordination of platelet agonist signaling during the hemostatic response in vivo.

Serum semaphorin 7A is associated with the risk of acute atherothrombotic stroke.

Perivascular adipose tissue modulates carotid plaque formation induced by disturbed flow in mice.

Efficient Optical Reflection Modulation by Coupling Interband Transition of Graphene to Magnetic Resonance in Metamaterials.

Humanin analogue, HNG, inhibits platelet activation and thrombus formation by stabilizing platelet microtubules.

Semaphorin 7A promotes endothelial to mesenchymal transition through ATF3 mediated TGF-β2/Smad signaling.

Molecular docking-assisted screening reveals tannic acid as a natural protein disulphide isomerase inhibitor with antiplatelet and antithrombotic activities.

Targeting Platelet in Atherosclerosis Plaque Formation: Current Knowledge and Future Perspectives.

Single-cell RNA-seq reveals cellular heterogeneity of mouse carotid artery under disturbed flow.

CLEC-2-dependent platelet subendothelial accumulation by flow disturbance contributes to atherogenesis in mice.

TET1s deficiency exacerbates oscillatory shear flow-induced atherosclerosis.

Single-cell transcriptional profiling of human carotid plaques reveals a subpopulation of endothelial cells associated with stroke incidences.

Sema7A protects against high-fat diet-induced obesity and hepatic steatosis by regulating adipo/lipogenesis.

The role of blood flow in vessel remodeling and its regulatory mechanism during developmental angiogenesis.

Endothelial CCRL2 induced by disturbed flow promotes atherosclerosis via chemerin-dependent β2 integrin activation in monocytes.

Corrigendum to "Sema7A protects against high-fat diet-induced obesity and hepatic steatosis by regulating adipo/lipogenesis" [Mol Metab. 70 (2023) 101698]∗.