Anjali M. Rajadhyaksha

Psychostimulants, L-type calcium channels, kinases, and phosphatases.

Psychostimulants, Protein phosphorylation and Gene expression: a growing role of L-type calcium channels.

NMDA receptors in the rat VTA: a critical site for social stress to intensify cocaine taking.

Dysregulation of large-conductance Ca2+-activated K+ channel expression in nonsyndromal mental retardation due to a cereblon p.R419X mutation.

Enhanced CREB and DARPP-32 phosphorylation in the nucleus accumbens and CREB, ERK, and GluR1 phosphorylation in the dorsal hippocampus is associated with cocaine-conditioned place preference behavior.

Deletion of the glutamate receptor 5 subunit of kainate receptors affects the development of morphine tolerance.

Deletion of the GluR5 subunit of kainate receptors affects cocaine sensitivity and preference.

Temporal and spatial mouse brain expression of cereblon, an ionic channel regulator involved in human intelligence.

Prenatal L-DOPA exposure produces lasting changes in brain dopamine content, cocaine-induced dopamine release and cocaine conditioned place preference.

Mutations in FLVCR1 cause posterior column ataxia and retinitis pigmentosa.

Molecular switch from L-type Ca v 1.3 to Ca v 1.2 Ca2+ channel signaling underlies long-term psychostimulant-induced behavioral and molecular plasticity.

Cav1.2 L-type Ca²⁺ channels mediate cocaine-induced GluA1 trafficking in the nucleus accumbens, a long-term adaptation dependent on ventral tegmental area Ca(v)1.3 channels.

Behavioral characterization of cereblon forebrain-specific conditional null mice: a model for human non-syndromic intellectual disability.

Enhanced dopamine D1 and BDNF signaling in the adult dorsal striatum but not nucleus accumbens of prenatal cocaine treated mice.

Brain-derived neurotrophic factor genotype impacts the prenatal cocaine-induced mouse phenotype.

Forebrain elimination of cacna1c mediates anxiety-like behavior in mice.

Serologic markers of Lyme disease in children with autism.

Markers of Celiac Disease and Gluten Sensitivity in Children with Autism.