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Myology Research Center,
UM76-INSERM U974-CNRS FRE 361,
Myology Research Center, UM76-INSERM U974-CNRS FRE 361
Bruno Cadot has not added Biography.
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Transglutaminase 5 is regulated by guanine-adenine nucleotides.
The Biochemical journal Jul, 2004 | Pubmed ID: 15038793
p73-alpha is capable of inducing scotin and ER stress.
Oncogene Apr, 2004 | Pubmed ID: 15116103
NMR structure of the p63 SAM domain and dynamical properties of G534V and T537P pathological mutants, identified in the AEC syndrome.
Cell biochemistry and biophysics , 2006 | Pubmed ID: 16679535
Loss of histone deacetylase 4 causes segregation defects during mitosis of p53-deficient human tumor cells.
Cancer research Aug, 2009 | Pubmed ID: 19622775
Identification and characterization of a non-satellite cell muscle resident progenitor during postnatal development.
Nature cell biology Mar, 2010 | Pubmed ID: 20118923
The sterile alpha-motif (SAM) domain of p63 binds in vitro monoasialoganglioside (GM1) micelles.
Biochemical pharmacology Nov, 2011 | Pubmed ID: 21820419
MAP and kinesin-dependent nuclear positioning is required for skeletal muscle function.
Nature Mar, 2012 | Pubmed ID: 22425998
Nuclear movement during myotube formation is microtubule and dynein dependent and is regulated by Cdc42, Par6 and Par3.
EMBO reports Aug, 2012 | Pubmed ID: 22732842
[Spatial distribution of nuclei in muscle fibers: a novel actor of muscular function].
Medecine sciences : M/S Jun-Jul, 2012 | Pubmed ID: 22805130
Fast, multi-dimensional and simultaneous kymograph-like particle dynamics (SkyPad) analysis.
PloS one , 2014 | Pubmed ID: 24586511
Pax3 and Pax7 play essential safeguard functions against environmental stress-induced birth defects.
Developmental cell Apr, 2015 | Pubmed ID: 25800090
Moving and positioning the nucleus in skeletal muscle - one step at a time.
Nucleus (Austin, Tex.) , 2015 | Pubmed ID: 26338260
A system for studying mechanisms of neuromuscular junction development and maintenance.
Development (Cambridge, England) Jul, 2016 | Pubmed ID: 27226316
Dynein disruption perturbs post-synaptic components and contributes to impaired MuSK clustering at the NMJ: implication in ALS.
Scientific reports Jun, 2016 | Pubmed ID: 27283349
Sorbonne University, UPMC University of Paris 6
Mafalda R. Pimentel1,
Sestina Falcone2,
Bruno Cadot2,
Edgar R. Gomes1,2
1Instituto de Medicina Molecular, Faculdade de Medicina, Universidade de Lisboa,
2Myology Research Center, UM76-INSERM U974-CNRS FRE 361, Sorbonne University, UPMC University of Paris 6
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