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BAD, a proapoptotic member of the BCL2 family, is a potential therapeutic target in hepatocellular carcinoma.
Molecular cancer research : MCR Aug, 2010 | Pubmed ID: 20647330
[Regulation of cell survival by RAF kinases].
Medecine sciences : M/S Aug-Sep, 2010 | Pubmed ID: 20819710
The Bcl-2 homology domain 3 (BH3) mimetic ABT-737 reveals the dynamic regulation of bad, a proapoptotic protein of the Bcl-2 family, by Bcl-xL.
Molecular pharmacology Jun, 2011 | Pubmed ID: 21393385
EGFR activation is a potential determinant of primary resistance of hepatocellular carcinoma cells to sorafenib.
International journal of cancer Dec, 2012 | Pubmed ID: 22514082
Iron-dependent cell death of hepatocellular carcinoma cells exposed to sorafenib.
International journal of cancer Oct, 2013 | Pubmed ID: 23505071
Heterogeneous sensitivity of hepatocellular carcinoma to sorafenib revealed by the short-term culture of tumor fragments.
Anticancer research Apr, 2013 | Pubmed ID: 23564781
[Ferroptosis, a new form of cell death relevant to the medical treatment of cancer].
Medecine sciences : M/S Aug-Sep, 2014 | Pubmed ID: 25174755
The microenvironment controls invadosome plasticity.
Journal of cell science May, 2016 | Pubmed ID: 27029343
TGF-β1 promotes linear invadosome formation in hepatocellular carcinoma cells, through DDR1 up-regulation and collagen I cross-linking.
European journal of cell biology Nov, 2016 | Pubmed ID: 27720259
Unr defines a novel class of nucleoplasmic reticulum involved in mRNA translation.
Journal of cell science May, 2017 | Pubmed ID: 28386023