Steven L. Bealer

Central Neuronal Histamine Contributes to Cardiovascular Regulation.

Systemic angiotensin II alters intrinsic heart rate through central mechanisms.

Increased dietary sodium alters neural control of blood pressure during intravenous ANG II infusion.

Peripheral hyperosmolality reduces cardiac baroreflex sensitivity.

Angiotensin II-induced release of oxytocin: interaction with norepinephrine and role in lactation.

Central neurokinin 3 receptors increase systemic oxytocin release: interaction with norepinephrine.

Differential sensitivity of intranuclear and systemic oxytocin release to central noradrenergic receptor stimulation during mid- and late gestation in rats.

Increased dietary sodium enhances activation of neurons in the medullary cardiovascular pathway during acute sodium loading in the rat.

Increased dietary sodium inhibits baroreflex-induced bradycardia during acute sodium loading.

Oxytocin receptor binding in the hypothalamus during gestation in rats.

Increased dietary sodium alters Fos expression in the lamina terminalis during intravenous angiotensin II infusion.

Status epilepticus produces chronic alterations in cardiac sympathovagal balance.

Status epilepticus induces cardiac myofilament damage and increased susceptibility to arrhythmias in rats.

Oxytocin release in magnocellular nuclei: neurochemical mediators and functional significance during gestation.

Autonomic and cellular mechanisms mediating detrimental cardiac effects of status epilepticus.

Beta adrenergic blockade prevents cardiac dysfunction following status epilepticus in rats.