Yu-Ching Su

Vitronectin in bacterial pathogenesis: a host protein used in complement escape and cellular invasion.

Moraxella catarrhalis: from interactions with the host immune system to vaccine development.

Haemophilus influenzae acquires vitronectin via the ubiquitous Protein F to subvert host innate immunity.

Impact of sequence diversity in the Moraxella catarrhalis UspA2/UspA2H head domain on vitronectin binding and antigenic variation.

Complete Genome Sequence of Encapsulated Haemophilus influenzae Type f KR494, an Invasive Isolate That Caused Necrotizing Myositis.

Comparative genomic analysis reveals distinct genotypic features of the emerging pathogen Haemophilus influenzae type f.

Impact of immunization with Protein F on pulmonary clearance of nontypeable Haemophilus influenzae.

A fine-tuned interaction between trimeric autotransporter haemophilus surface fibrils and vitronectin leads to serum resistance and adherence to respiratory epithelial cells.

Outer membrane protein OlpA contributes to Moraxella catarrhalis serum resistance via interaction with factor H and the alternative pathway.

Identification of a Haemophilus influenzae factor H-Binding lipoprotein involved in serum resistance.

Identification of outer membrane Porin D as a vitronectin-binding factor in cystic fibrosis clinical isolates of Pseudomonas aeruginosa.

Moraxella catarrhalis Binds Plasminogen To Evade Host Innate Immunity.

Haemophilus influenzae P4 Interacts With Extracellular Matrix Proteins Promoting Adhesion and Serum Resistance.

Haemophilus influenzae Type f Hijacks Vitronectin Using Protein H To Resist Host Innate Immunity and Adhere to Pulmonary Epithelial Cells.

Moraxella catarrhalis Evades Host Innate Immunity via Targeting Cartilage Oligomeric Matrix Protein.

Moonlighting of Helicobacter pylori catalase protects against complement-mediated killing by utilising the host molecule vitronectin.

Host-pathogen interactions of nontypeable Haemophilus influenzae: from commensal to pathogen.

PRELP Enhances Host Innate Immunity against the Respiratory Tract Pathogen .

Haemophilus Protein F Orthologs of Pathogens Infecting the Airways: Exploiting Host Laminin at Heparin-Binding Sites for Maximal Adherence to Epithelial Cells.

A novel PBP3 substitution in Haemophilus influenzae confers reduced aminopenicillin susceptibility.

Bacterial Outer Membrane Vesicles Induce Vitronectin Release Into the Bronchoalveolar Space Conferring Protection From Complement-Mediated Killing.

Outer Membrane Vesicles Protect the Pathogen From Reactive Oxygen Species of the Respiratory Burst.