Zsolt Bagi

Xanthine oxidase-derived reactive oxygen species convert flow-induced arteriolar dilation to constriction in hyperhomocysteinemia: possible role of peroxynitrite.

New trends in the development of oral antidiabetic drugs.

Impaired nitric oxide-mediated flow-induced coronary dilation in hyperhomocysteinemia: morphological and functional evidence for increased peroxynitrite formation.

Electrophysiological effects of risperidone in mammalian cardiac cells.

On the role of mechanosensitive mechanisms eliciting reactive hyperemia.

Lack of nitric oxide mediation of flow-dependent arteriolar dilation in type I diabetes is restored by sepiapterin.

Chronic renal failure leads to reduced flow-dependent dilation in isolated rat skeletal muscle arterioles due to lack of NO mediation.

Superoxide-NO interaction decreases flow- and agonist-induced dilations of coronary arterioles in Type 2 diabetes mellitus.

Oxidative stress-induced dysregulation of arteriolar wall shear stress and blood pressure in hyperhomocysteinemia is prevented by chronic vitamin C treatment.

PPARgamma activation, by reducing oxidative stress, increases NO bioavailability in coronary arterioles of mice with Type 2 diabetes.

Microvascular dysfunction after transient high glucose is caused by superoxide-dependent reduction in the bioavailability of NO and BH(4).

Ghrelin induces vasoconstriction in the rat coronary vasculature without altering cardiac peptide secretion.

Biphasic effect of hydrogen peroxide on skeletal muscle arteriolar tone via activation of endothelial and smooth muscle signaling pathways.

Nitric oxide and H2O2 contribute to reactive dilation of isolated coronary arterioles.

Expression and distribution of vanilloid receptor 1 (TRPV1) in the adult rat brain.

PECAM-1 mediates NO-dependent dilation of arterioles to high temporal gradients of shear stress.

Type 2 diabetic mice have increased arteriolar tone and blood pressure: enhanced release of COX-2-derived constrictor prostaglandins.

Phosphorylation-dependent desensitization by anandamide of vanilloid receptor-1 (TRPV1) function in rat skeletal muscle arterioles and in Chinese hamster ovary cells expressing TRPV1.

The levosimendan metabolite OR-1896 elicits vasodilation by activating the K(ATP) and BK(Ca) channels in rat isolated arterioles.

High-fat diet-induced reduction in nitric oxide-dependent arteriolar dilation in rats: role of xanthine oxidase-derived superoxide anion.

Increased cyclooxygenase-2 expression and prostaglandin-mediated dilation in coronary arterioles of patients with diabetes mellitus.

H2O2 increases production of constrictor prostaglandins in smooth muscle leading to enhanced arteriolar tone in Type 2 diabetic mice.

Activation of the poly(ADP-ribose) polymerase pathway in human heart failure.

Asymmetrical dimethylarginine inhibits shear stress-induced nitric oxide release and dilation and elicits superoxide-mediated increase in arteriolar tone.

Up-regulation of vascular cyclooxygenase-2 in diabetes mellitus.

Adaptation of vasomotor function of human coronary arterioles to the simultaneous presence of obesity and hypertension.

Contribution of polyol pathway to arteriolar dysfunction in hyperglycemia. Role of oxidative stress, reduced NO, and enhanced PGH(2)/TXA(2) mediation.

Tissue-specific regulation of microvascular diameter: opposite functional roles of neuronal and smooth muscle located vanilloid receptor-1.

High-fat diet-induced obesity leads to increased NO sensitivity of rat coronary arterioles: role of soluble guanylate cyclase activation.

High intraluminal pressure via H2O2 upregulates arteriolar constrictions to angiotensin II by increasing the functional availability of AT1 receptors.

Where have all the stem cells gone?

Activation of prostaglandin E2 EP1 receptor increases arteriolar tone and blood pressure in mice with type 2 diabetes.

Preserved coronary arteriolar dilatation in patients with type 2 diabetes mellitus: implications for reactive oxygen species.

Mechanisms of coronary microvascular adaptation to obesity.

Cyclooxygenase-2 derived thromboxane A(2) and reactive oxygen species mediate flow-induced constrictions of venules in hyperhomocysteinemia.

Resveratrol attenuates mitochondrial oxidative stress in coronary arterial endothelial cells.

Reactive oxygen species and cyclooxygenase 2-derived thromboxane A2 reduce angiotensin II type 2 receptor vasorelaxation in diabetic rat resistance arteries.

Caveolin-1 limits the contribution of BK(Ca) channel to EDHF-mediated arteriolar dilation: implications in diet-induced obesity.

Resveratrol confers endothelial protection via activation of the antioxidant transcription factor Nrf2.

Hypoxic relaxation of penile arteries: involvement of endothelial nitric oxide and modulation by reactive oxygen species.

Arginase 1 contributes to diminished coronary arteriolar dilation in patients with diabetes.

Increased availability of angiotensin AT 1 receptors leads to sustained arterial constriction to angiotensin II in diabetes - role for Rho-kinase activation.

Microvascular responsiveness in obesity: implications for therapeutic intervention.

Molecular imaging with optical coherence tomography using ligand-conjugated microparticles that detect activated endothelial cells: rational design through target quantification.

Activation of hexosamine pathway impairs nitric oxide (NO)-dependent arteriolar dilations by increased protein O-GlcNAcylation.

Exacerbation of endothelial dysfunction during the progression of diabetes: role of oxidative stress.

Endothelial regulation of coronary microcirculation in health and cardiometabolic diseases.

Increased tissue angiotensin-converting enzyme activity impairs bradykinin-induced dilation of coronary arterioles in obesity.

Obesity and statins are both independent predictors of enhanced coronary arteriolar dilation in patients undergoing heart surgery.

Selective up-regulation of arginase-1 in coronary arteries of diabetic patients.

Effect of a novel stobadine derivative on isolated rat arteries.

Peroxynitrite disrupts endothelial caveolae leading to eNOS uncoupling and diminished flow-mediated dilation in coronary arterioles of diabetic patients.

Levosimendan and its metabolite OR-1896 elicit KATP channel-dependent dilation in resistance arteries in vivo.

l-Citrulline Protects from Kidney Damage in Type 1 Diabetic Mice.

Prevention and treatment of no-reflow phenomenon by targeting the coronary microcirculation.

Age-related impairment of conducted dilation in human coronary arterioles.

Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.

Obesity and coronary microvascular disease - implications for adipose tissue-mediated remote inflammatory response.

A novel tumor necrosis factor-mediated mechanism of direct epithelial sodium channel activation.

Caveolin-1 prevents sustained angiotensin II-induced resistance artery constriction and obesity-induced high blood pressure.

Impaired coronary collateral growth: miR-shaken neutrophils caught in the act.