Nadia Rosenthal

Different modes of hypertrophy in skeletal muscle fibers.

Targeted deletion of the MLC1f/3f downstream enhancer results in precocious MLC expression and mesoderm ablation.

Muscle-specific expression of insulin-like growth factor I counters muscle decline in mdx mice.

The role of stem cells in skeletal and cardiac muscle repair.

Gene therapy for cardiac cachexia?

Taking stock of our models: the function and future of stock centres.

Machinations of the marrow.

Normal female sexual development requires neuregulin-erbB receptor signaling in hypothalamic astrocytes.

Prometheus's vulture and the stem-cell promise.

Developmental basis of evolutionary digit loss in the Australian lizard Hemiergis.

A caudorostral wave of RALDH2 conveys anteroposterior information to the cardiac field.

Stem cell-mediated muscle regeneration is enhanced by local isoform of insulin-like growth factor 1.

The stem cell challenge.

The European dimension for the mouse genome mutagenesis program.

Targeted expression of insulin-like growth factor-I reduces early myofiber necrosis in dystrophic mdx mice.

Human hepatocytes in mice receiving pre-immune injection with human cord blood cells.

Muscle-specific expression of IGF-1 blocks angiotensin II-induced skeletal muscle wasting.

Muscle expression of a local Igf-1 isoform protects motor neurons in an ALS mouse model.

Reconciling data from transgenic mice that overexpress IGF-I specifically in skeletal muscle.

Insulin-like growth factor I-mediated skeletal muscle hypertrophy is characterized by increased mTOR-p70S6K signaling without increased Akt phosphorylation.

Vasopressin-dependent myogenic cell differentiation is mediated by both Ca2+/calmodulin-dependent kinase and calcineurin pathways.

Youthful prospects for human stem-cell therapy. In another few decades, revised attitudes toward stem cells could lead to disease prevention and life extension.

Transgenic overexpression of locally acting insulin-like growth factor-1 inhibits ubiquitin-mediated muscle atrophy in chronic left-ventricular dysfunction.

IGF-1, inflammation and stem cells: interactions during muscle regeneration.

The future of regenerative therapy in patients with chronic heart failure.

The neuroprotective effects of a locally acting IGF-1 isoform.

Growth factor enhancement of cardiac regeneration.

Signalling pathways in cardiac regeneration.

Targeted ablation of IKK2 improves skeletal muscle strength, maintains mass, and promotes regeneration.

Local expression of IGF-1 accelerates muscle regeneration by rapidly modulating inflammatory cytokines and chemokines.

Identification of novel peptide hormones in the human proteome by hidden Markov model screening.

Enhancing repair of the mammalian heart.

Immune response to stem cells and strategies to induce tolerance.

ROCK2 and its alternatively spliced isoform ROCK2m positively control the maturation of the myogenic program.

Phenostat: visualization and statistical tool for analysis of phenotyping data.

The mouse ascending: perspectives for human-disease models.

Derive and conquer: sourcing and differentiating stem cells for therapeutic applications.

A naturally occurring calcineurin variant inhibits FoxO activity and enhances skeletal muscle regeneration.

NF-kappaB signaling in skeletal muscle: prospects for intervention in muscle diseases.

Reliability, robustness, and reproducibility in mouse behavioral phenotyping: a cross-laboratory study.

Interleukin-10 from transplanted bone marrow mononuclear cells contributes to cardiac protection after myocardial infarction.

Expression of follistatin-related genes is altered in heart failure.

Analysis of CRE-mediated recombination driven by myosin light chain 1/3 regulatory elements in embryonic and adult skeletal muscle: a tool to study fiber specification.

Overexpression of mIGF-1 in keratinocytes improves wound healing and accelerates hair follicle formation and cycling in mice.

Skeletal muscle is a primary target of SOD1G93A-mediated toxicity.

Multiple congenital malformations of Wolf-Hirschhorn syndrome are recapitulated in Fgfrl1 null mice.

Post-publication sharing of data and tools.

A CREB-C/EBPbeta cascade induces M2 macrophage-specific gene expression and promotes muscle injury repair.

Distinct roles for cell-autonomous Notch signaling in cardiomyocytes of the embryonic and adult heart.

Insights into the organization of dorsal spinal cord pathways from an evolutionarily conserved raldh2 intronic enhancer.

Models for financial sustainability of biological databases and resources.

A growth stimulus is needed for IGF-1 to induce skeletal muscle hypertrophy in vivo.

Analysis of cardiac myocyte biology in transgenic mice: a protocol for preparation of neonatal mouse cardiac myocyte cultures.

Local IGF-1 isoform protects cardiomyocytes from hypertrophic and oxidative stresses via SirT1 activity.

SirT1 in muscle physiology and disease: lessons from mouse models.

Comments on Point:Counterpoint: IGF is/is not the major physiological regulator of muscle mass. IGF-1 is a major regulator of muscle mass during growth but not for adult myofiber hypertrophy.

Serum and glucocorticoid-inducible kinase 1 (SGK1) is necessary for vascular remodeling during angiogenesis.

Revealing new mouse epicardial cell markers through transcriptomics.

Mouse Resource Browser--a database of mouse databases.

Finding and sharing: new approaches to registries of databases and services for the biomedical sciences.

Regulation of muscle atrophy in aging and disease.

Research funding. Sustaining the data and bioresource commons.

Cre recombinase resources for conditional mouse mutagenesis.

Insulin-like growth factor-1 overexpression in cardiomyocytes diminishes ex vivo heart functional recovery after acute ischemia.

IGF-1Ea induces vessel formation after injury and mediates bone marrow and heart cross-talk through the expression of specific cytokines.

Calcineurin splicing variant calcineurin Aβ1 improves cardiac function after myocardial infarction without inducing hypertrophy.

MicroRNA-29 in aortic dilation: implications for aneurysm formation.

Complex SUMO-1 regulation of cardiac transcription factor Nkx2-5.

mIGF-1/JNK1/SirT1 signaling confers protection against oxidative stress in the heart.

Increased cardiogenesis in P19-GFP teratocarcinoma cells expressing the propeptide IGF-1Ea.

Proximal tubule overexpression of a locally acting IGF isoform, Igf-1Ea, increases inflammation after ischemic injury.

In search of a wide-angle perspective.

Locally expressed IGF1 propeptide improves mouse heart function in induced dilated cardiomyopathy by blocking myocardial fibrosis and SRF-dependent CTGF induction.

An abundant tissue macrophage population in the adult murine heart with a distinct alternatively-activated macrophage profile.

Myocardial regenerative properties of macrophage populations and stem cells.

CreZOO--the European virtual repository of Cre and other targeted conditional driver strains.

Follistatin-like 3 mediates paracrine fibroblast activation by cardiomyocytes.

Beyond knockouts: cre resources for conditional mutagenesis.

The mammalian gene function resource: the International Knockout Mouse Consortium.

E-peptides control bioavailability of IGF-1.

Nerve growth factor stimulates cardiac regeneration via cardiomyocyte proliferation in experimental heart failure.

Isolation and analysis of single cells from the mouse heart.

Functional Characterization of a Novel Mutation in NKX2-5 Associated with Congenital Heart Disease and Adult-Onset Cardiomyopathy.

Macrophages are required for adult salamander limb regeneration.

Cardiac fibrosis in mice expressing an inducible myocardial-specific Cre driver.

Ablation of SGK1 impairs endothelial cell migration and tube formation leading to decreased neo-angiogenesis following myocardial infarction.