Laurent Brossay

Cutting edge: inhibitory functions of the killer cell lectin-like receptor G1 molecule during the activation of mouse NK cells.

NK cell receptors: emerging roles in host defense against infectious agents.

Autoreactive T cells can be protected from tolerance induction through competition by flanking determinants for access to class II MHC.

NK T cell-NK cell cross-talk: reciprocal interaction and activation?

Differential regulation of killer cell lectin-like receptor G1 expression on T cells.

Inhibition of CD1d activation suppresses septic mortality: a role for NK-T cells in septic immune dysfunction.

Differential distribution of the JC virus receptor-type sialic acid in normal human tissues.

Expansion and contraction of the NK cell compartment in response to murine cytomegalovirus infection.

Expansion and function of CD8+ T cells expressing Ly49 inhibitory receptors specific for MHC class I molecules.

Direct effects of T-bet and MHC class I expression, but not STAT1, on peripheral NK cell maturation.

The mouse CD1d cytoplasmic tail mediates CD1d trafficking and antigen presentation by adaptor protein 3-dependent and -independent mechanisms.

Cutting edge: IFN-gamma signaling to macrophages is required for optimal Valpha14i NK T/NK cell cross-talk.

KLRG1 binds cadherins and preferentially associates with SHIP-1.

A Y chromosome-linked factor impairs NK T development.

The ontogeny and fate of NK cells marked by permanent DNA rearrangements.

A role for interleukin-12/23 in the maturation of human natural killer and CD56+ T cells in vivo.

NK cell-like behavior of Valpha14i NK T cells during MCMV infection.

Cutting edge: Priming of NK cells by IL-18.

Invariant natural killer T cells suppress the neutrophil inflammatory response in a mouse model of cholestatic liver damage.

NKT cell immune responses to viral infection.

A new approach for quantitative phosphoproteomic dissection of signaling pathways applied to T cell receptor activation.

Immunoregulatory functions of KLRG1 cadherin interactions are dependent on forward and reverse signaling.

Cytokine dependent and independent iNKT cell activation.

Salivary gland NK cells are phenotypically and functionally unique.

Comparing the kinetics of NK cells, CD4, and CD8 T cells in murine cytomegalovirus infection.