Norishige Yamada

Nerve damage in Mycobacterium ulcerans-infected mice: probable cause of painlessness in buruli ulcer.

MUC2 expression is regulated by histone H3 modification and DNA methylation in pancreatic cancer.

MUC1 expression is regulated by DNA methylation and histone H3 lysine 9 modification in cancer cells.

Expression profiles of MUC1, MUC2, and MUC4 mucins in human neoplasms and their relationship with biological behavior.

Significance of mucin expression in pancreatobiliary neoplasms.

Precursor lesions of pancreatic cancer.

Promoter hypomethylation contributes to the expression of MUC3A in cancer cells.

Expression of MUC5AC, an early marker of pancreatobiliary cancer, is regulated by DNA methylation in the distal promoter region in cancer cells.

DNA methylation and histone H3-K9 modifications contribute to MUC17 expression.

MUC4: a novel prognostic factor of oral squamous cell carcinoma.

Comprehensive epigenetic analysis using oral rinse samples: a pilot study.

Mucins in human neoplasms: clinical pathology, gene expression and diagnostic application.

A novel anti-MUC1 antibody against the MUC1 cytoplasmic tail domain: use in sensitive identification of poorly differentiated cells in adenocarcinoma of the stomach.

Aberrant DNA methylation of tumor-related genes in oral rinse: a noninvasive method for detection of oral squamous cell carcinoma.

Pathobiological implications of MUC16/CA125 expression in intrahepatic cholangiocarcinoma-mass forming type.

The application of methylation specific electrophoresis (MSE) to DNA methylation analysis of the 5' CpG island of mucin in cancer cells.

DF3 epitope expression on MUC1 mucin is associated with tumor aggressiveness, subsequent lymph node metastasis, and poor prognosis in patients with oral squamous cell carcinoma.

Epigenetic regulation of mucin genes in human cancers.

Expression of MUC17 is regulated by HIF1α-mediated hypoxic responses and requires a methylation-free hypoxia responsible element in pancreatic cancer.

CCAAT/enhancer-binding protein-α suppresses lung tumor development in mice through the p38α MAP kinase pathway.

Combination of MUC1 and MUC4 expression predicts clinical outcome in patients with oral squamous cell carcinoma.