Brenda Cordeiro

p38 MAPK-dependent small HSP27 and αB-crystallin phosphorylation in regulation of myocardial function following cardioplegic arrest.

Rottlerin increases cardiac contractile performance and coronary perfusion through BKCa++ channel activation after cold cardioplegic arrest in isolated hearts.

Oxidative stress improves coronary endothelial function through activation of the pro-survival kinase AMPK.

Rottlerin-induced BKCa channel activation impairs specific contractile responses and promotes vasodilation.

BKCa channel activation increases cardiac contractile recovery following hypothermic ischemia / reperfusion.