Mehrnoosh Saghizadeh

Evaluation of techniques using amplified nucleic acid probes for gene expression profiling.

Increased levels of catalase and cathepsin V/L2 but decreased TIMP-1 in keratoconus corneas: evidence that oxidative stress plays a role in this disorder.

Proteinase and growth factor alterations revealed by gene microarray analysis of human diabetic corneas.

Expression of protein kinase CK2 in astroglial cells of normal and neovascularized retina.

Identification and characterization of genes expressed in cone photoreceptors.

Alterations of extracellular matrix components and proteinases in human corneal buttons with INTACS for post-laser in situ keratomileusis keratectasia and keratoconus.

ZBED4, a BED-type zinc-finger protein in the cones of the human retina.

Adenovirus-driven overexpression of proteinases in organ-cultured normal human corneas leads to diabetic-like changes.

Normalization of wound healing and diabetic markers in organ cultured human diabetic corneas by adenoviral delivery of c-Met gene.

ZBED4, a cone and Müller cell protein in human retina, has a different cellular expression in mouse.

Alterations of epithelial stem cell marker patterns in human diabetic corneas and effects of c-met gene therapy.

A simple alkaline method for decellularizing human amniotic membrane for cell culture.

Enhanced wound healing, kinase and stem cell marker expression in diabetic organ-cultured human corneas upon MMP-10 and cathepsin F gene silencing.

Differentially expressed wound healing-related microRNAs in the human diabetic cornea.

Differentiation of human limbal-derived induced pluripotent stem cells into limbal-like epithelium.

Normalization of wound healing and stem cell marker patterns in organ-cultured human diabetic corneas by gene therapy of limbal cells.

Targeting miR-146a to treat delayed wound healing in human diabetic organ-cultured corneas.

Progress in corneal wound healing.

Persistence of reduced expression of putative stem cell markers and slow wound healing in cultured diabetic limbal epithelial cells.