Xuelin Lou

Protein kinase activation increases insulin secretion by sensitizing the secretory machinery to Ca2+.

Presynaptic Ca2+ requirements and developmental regulation of posttetanic potentiation at the calyx of Held.

Allosteric modulation of the presynaptic Ca2+ sensor for vesicle fusion.

A Munc13/RIM/Rab3 tripartite complex: from priming to plasticity?

A mechanism intrinsic to the vesicle fusion machinery determines fast and slow transmitter release at a large CNS synapse.

Posttetanic potentiation critically depends on an enhanced Ca(2+) sensitivity of vesicle fusion mediated by presynaptic PKC.

Phorbol esters modulate spontaneous and Ca2+-evoked transmitter release via acting on both Munc13 and protein kinase C.

Selective saturation of slow endocytosis at a giant glutamatergic central synapse lacking dynamin 1.

Regulation of transmitter release by Ca(2+) and synaptotagmin: insights from a large CNS synapse.

Overlapping role of dynamin isoforms in synaptic vesicle endocytosis.

Recruitment of endophilin to clathrin-coated pit necks is required for efficient vesicle uncoating after fission.

Reduced release probability prevents vesicle depletion and transmission failure at dynamin mutant synapses.

Nanoscale Landscape of Phosphoinositides Revealed by Specific Pleckstrin Homology (PH) Domains Using Single-molecule Superresolution Imaging in the Plasma Membrane.

Dynamin 2 regulates biphasic insulin secretion and plasma glucose homeostasis.

Tissue-specific dynamin-1 deletion at the calyx of Held decreases short-term depression through a mechanism distinct from vesicle resupply.

Dynamin-1 deletion enhances post-tetanic potentiation and quantal size after tetanic stimulation at the calyx of Held.

Dynamin 1- and 3-Mediated Endocytosis Is Essential for the Development of a Large Central Synapse In Vivo.