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Animal Surgery and Resources Core,
National Heart,
Lung,
and Blood Institute,
Animal Surgery and Resources Core, National Heart, Lung, and Blood Institute
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Apolipoprotein E negatively regulates house dust mite-induced asthma via a low-density lipoprotein receptor-mediated pathway.
American journal of respiratory and critical care medicine Nov, 2010 | Pubmed ID: 20622028
5A, an apolipoprotein A-I mimetic peptide, attenuates the induction of house dust mite-induced asthma.
Journal of immunology (Baltimore, Md. : 1950) Jan, 2011 | Pubmed ID: 21115733
Human apolipoprotein E genotypes differentially modify house dust mite-induced airway disease in mice.
American journal of physiology. Lung cellular and molecular physiology Jan, 2012 | Pubmed ID: 22058162
Apolipoprotein A-I attenuates ovalbumin-induced neutrophilic airway inflammation via a granulocyte colony-stimulating factor-dependent mechanism.
American journal of respiratory cell and molecular biology Aug, 2012 | Pubmed ID: 22427535
Paradoxical effects of rapamycin on experimental house dust mite-induced asthma.
PloS one , 2012 | Pubmed ID: 22685525
Peptidoglycan recognition protein 1 promotes house dust mite-induced airway inflammation in mice.
American journal of respiratory cell and molecular biology Dec, 2013 | Pubmed ID: 23808363
The very low density lipoprotein receptor attenuates house dust mite-induced airway inflammation by suppressing dendritic cell-mediated adaptive immune responses.
Journal of immunology (Baltimore, Md. : 1950) May, 2014 | Pubmed ID: 24733846
ATP-binding cassette transporter 1 attenuates ovalbumin-induced neutrophilic airway inflammation.
American journal of respiratory cell and molecular biology Nov, 2014 | Pubmed ID: 24813055
Dendritic cells induce Th2-mediated airway inflammatory responses to house dust mite via DNA-dependent protein kinase.
Nature communications Feb, 2015 | Pubmed ID: 25692509
National Institutes of Health
Karen J. Keeran*,1,
Kenneth R. Jeffries1,
Arthur D. Zetts1,
Joni Taylor1,
Shawn Kozlov1,
Timothy J. Hunt*,1
1Animal Surgery and Resources Core, National Heart, Lung, and Blood Institute, National Institutes of Health
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