Maria Bloksgaard

The gene encoding acyl-CoA-binding protein is subject to metabolic regulation by both sterol regulatory element-binding protein and peroxisome proliferator-activated receptor alpha in hepatocytes.

Disruption of the acyl-CoA-binding protein gene delays hepatic adaptation to metabolic changes at weaning.

Mice with targeted disruption of the acyl-CoA binding protein display attenuated urine concentrating ability and diminished renal aquaporin-3 abundance.

Structural characterization and lipid composition of acquired cholesteatoma: a comparative study with normal skin.

The acyl-CoA binding protein is required for normal epidermal barrier function in mice.

Spatially resolved two-color diffusion measurements in human skin applied to transdermal liposome penetration.

Structural and dynamical aspects of skin studied by multiphoton excitation fluorescence microscopy-based methods.

Acyl-CoA binding protein and epidermal barrier function.

Delayed hepatic adaptation to weaning in ACBP-/- mice is caused by disruption of the epidermal barrier.

Elastin organization in pig and cardiovascular disease patients' pericardial resistance arteries.

Compromised epidermal barrier stimulates Harderian gland activity and hypertrophy in ACBP-/- mice.

Endothelin-1 shifts the mediator of bradykinin-induced relaxation from NO to H2 O2 in resistance arteries from patients with cardiovascular disease.

Biochemical and Bioimaging Evidence of Cholesterol in Acquired Cholesteatoma.

Endothelial SIRT1 prevents adverse arterial remodeling by facilitating HERC2-mediated degradation of acetylated LKB1.

Imaging and modeling of acute pressure-induced changes of collagen and elastin microarchitectures in pig and human resistance arteries.

Relaxing Responses to Hydrogen Peroxide and Nitric Oxide in Human Pericardial Resistance Arteries Stimulated with Endothelin-1.