Kathrin Plath

Reprogrammed mouse fibroblasts differentiate into cells of the cardiovascular and hematopoietic lineages.

Role of the murine reprogramming factors in the induction of pluripotency.

Epigenetic reprogramming and induced pluripotency.

Post-translational regulation of Oct4 transcriptional activity.

Directed differentiation of human-induced pluripotent stem cells generates active motor neurons.

Induced pluripotent stem cells and embryonic stem cells are distinguished by gene expression signatures.

Signed weighted gene co-expression network analysis of transcriptional regulation in murine embryonic stem cells.

Genome-wide dynamics of replication timing revealed by in vitro models of mouse embryogenesis.

Molecular analyses of human induced pluripotent stem cells and embryonic stem cells.

Female human iPSCs retain an inactive X chromosome.

Mechanistic insights into reprogramming to induced pluripotency.

Reprogramming to pluripotency: stepwise resetting of the epigenetic landscape.

Progress in understanding reprogramming to the induced pluripotent state.

Small RNAs loom large during reprogramming.

Mediator coordinates PIC assembly with recruitment of CHD1.

Concise review: Pluripotency and the transcriptional inactivation of the female Mammalian X chromosome.

Characterization and therapeutic potential of induced pluripotent stem cell-derived cardiovascular progenitor cells.

Pluripotency re-centered around Esrrb.

Stem cells in the land of the rising sun: ISSCR 2012.

Mediator and SAGA have distinct roles in Pol II preinitiation complex assembly and function.

Pluripotency in 3D: genome organization in pluripotent cells.

Epigenetics of reprogramming to induced pluripotency.

The pluripotency factor-bound intron 1 of Xist is dispensable for X chromosome inactivation and reactivation in vitro and in vivo.

Proteomic and genomic approaches reveal critical functions of H3K9 methylation and heterochromatin protein-1γ in reprogramming to pluripotency.

Stage-specific regulation of reprogramming to induced pluripotent stem cells by Wnt signaling and T cell factor proteins.

The Xist lncRNA exploits three-dimensional genome architecture to spread across the X chromosome.

A mechanistic link between gene regulation and genome architecture in mammalian development.

The Mbd1-Atf7ip-Setdb1 pathway contributes to the maintenance of X chromosome inactivation.

X chromosome reactivation dynamics reveal stages of reprogramming to pluripotency.

The Xist lncRNA interacts directly with SHARP to silence transcription through HDAC3.

A high-throughput screen of inactive X chromosome reactivation identifies the enhancement of DNA demethylation by 5-aza-2'-dC upon inhibition of ribonucleotide reductase.

X chromosome reactivation in reprogramming and in development.

The "lnc" between 3D chromatin structure and X chromosome inactivation.

Transcriptome Encyclopedia of Early Human Development.

Glycolytic Metabolism Plays a Functional Role in Regulating Human Pluripotent Stem Cell State.

Human Embryonic Stem Cells Do Not Change Their X Inactivation Status during Differentiation.

Human Naive Pluripotent Stem Cells Model X Chromosome Dampening and X Inactivation.

Cooperative Binding of Transcription Factors Orchestrates Reprogramming.

Cbx3 maintains lineage specificity during neural differentiation.

Regulation of X-chromosome dosage compensation in human: mechanisms and model systems.

Analysis of cardiomyocyte clonal expansion during mouse heart development and injury.

High-Throughput Screening of a Luciferase Reporter of Gene Silencing on the Inactive X Chromosome.

X Chromosome Dosage Influences DNA Methylation Dynamics during Reprogramming to Mouse iPSCs.

Loss of MECP2 Leads to Activation of P53 and Neuronal Senescence.

The Role of Xist in X-Chromosome Dosage Compensation.