Scott Grieshaber

Determination of the physical environment within the Chlamydia trachomatis inclusion using ion-selective ratiometric probes.

Chlamydia trachomatis induces remodeling of the actin cytoskeleton during attachment and entry into HeLa cells.

Follicle-stimulating hormone-responsive cytoskeletal genes in rat granulosa cells: class I beta-tubulin, tropomyosin-4, and kinesin heavy chain.

Effects of ectopically expressed neuronal Wiskott-Aldrich syndrome protein domains on Rickettsia rickettsii actin-based motility.

Chlamydia trachomatis uses host cell dynein to traffic to the microtubule-organizing center in a p50 dynamitin-independent process.

Requirement for the Rac GTPase in Chlamydia trachomatis invasion of non-phagocytic cells.

Tyrosine phosphorylation of the chlamydial effector protein Tarp is species specific and not required for recruitment of actin.

A small RNA inhibits translation of the histone-like protein Hc1 in Chlamydia trachomatis.

Chlamydia trachomatis causes centrosomal defects resulting in chromosomal segregation abnormalities.

Rac interacts with Abi-1 and WAVE2 to promote an Arp2/3-dependent actin recruitment during chlamydial invasion.

Chlamydial entry involves TARP binding of guanine nucleotide exchange factors.

Tissue Distibution of Murine Muc19/Smgc Gene Products.

Chlamydia trachomatis infection causes mitotic spindle pole defects independently from its effects on centrosome amplification.