Daniel Czuriga

Rate of tension redevelopment is not modulated by sarcomere length in permeabilized human, murine, and porcine cardiomyocytes.

Protein kinase C contributes to the maintenance of contractile force in human ventricular cardiomyocytes.

Beneficial effects of SR33805 in failing myocardium.

Cell-to-cell variability in troponin I phosphorylation in a porcine model of pacing-induced heart failure.

How cardiomyocytes make the heart old.

Cellular mechanisms for diastolic dysfunction in the human heart.

Silent brain infarction--a review of recent observations.

ST-segment elevation followed by progressive widening of the QRS complex.

Myofilament protein carbonylation contributes to the contractile dysfunction in the infarcted LV region of mouse hearts.

Transtelephonic electrocardiography in the management of patients with acute coronary syndrome.

Elevated LDL-C combined with hypertension worsens subclinical vascular impairment and cognitive function.

Cardiac Resynchronization Therapy Relieves Intractable Angina Due to Exercise-Induced Left Bundle Branch Block Without Left Ventricular Systolic Dysfunction: A Detailed Case Study.

Influence of Hypertension, Alone and in Combination with Other Vascular Risk Factors on Cognition.

The transtelephonic electrocardiogram-based triage is an independent predictor of decreased hospital mortality in patients with ST-segment elevation myocardial infarction treated with primary percutaneous coronary intervention.

Hypertension-induced subclinical vascular and cognitive changes are reversible-An observational cohort study.

Three-dimensional evaluation of the spatial morphology of stented coronary artery segments in relation to restenosis.

Reply to letter: Reversibility of hypertension-induced subclinical vascular changes: Do the new ACC/AHA 2017 blood pressure guidelines and heart rate changes make a difference?

Advantages of prophylactic versus conventionally scheduled heart failure therapy in an experimental model of doxorubicin-induced cardiomyopathy.

The impact of hydrostatic pressure on the result of physiological measurements in various coronary segments.