Roberto Chiesa

Inhibition of IL-1β Signaling Normalizes NMDA-Dependent Neurotransmission and Reduces Seizure Susceptibility in a Mouse Model of Creutzfeldt-Jakob Disease.

Induction of a transmissible tau pathology by traumatic brain injury.

Cellular prion protein neither binds to alpha-synuclein oligomers nor mediates their detrimental effects.

Experimental models to study prion disease pathogenesis and identify potential therapeutic compounds.

Age at onset in genetic prion disease and the design of preventive clinical trials.

Cyclophillin A deficiency accelerates RML-induced prion disease.

Editorial overview: Prion disease: From drug discovery tools to clinical trials.

Super Resolution Microscopy of SUMO Proteins in Neurons.

Calcineurin Controls Expression of EAAT1/GLAST in Mouse and Human Cultured Astrocytes through Dynamic Regulation of Protein Synthesis and Degradation.

Mutant prion proteins increase calcium permeability of AMPA receptors, exacerbating excitotoxicity.

Sleep inhibition induced by amyloid-β oligomers is mediated by the cellular prion protein.