Huan Yang

High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes.

Globin attenuates the innate immune response to endotoxin.

Bacterial endotoxin stimulates macrophages to release HMGB1 partly through CD14- and TNF-dependent mechanisms.

Suppression of HMGB1 release by stearoyl lysophosphatidylcholine:an additional mechanism for its therapeutic effects in experimental sepsis.

The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion.

The aqueous extract of a popular herbal nutrient supplement, Angelica sinensis, protects mice against lethal endotoxemia and sepsis.

A cardiovascular drug rescues mice from lethal sepsis by selectively attenuating a late-acting proinflammatory mediator, high mobility group box 1.

A major ingredient of green tea rescues mice from lethal sepsis partly by inhibiting HMGB1.

Spermine protects mice against lethal sepsis partly by attenuating surrogate inflammatory markers.

Peripheral administration of fetuin-A attenuates early cerebral ischemic injury in rats.

An Engineered orco Mutation Produces Aberrant Social Behavior and Defective Neural Development in Ants.

Social support modifies an association between work stress and semen quality: Results from 384 Chinese male workers.

Enhanced Macrophage Pannexin 1 Expression and Hemichannel Activation Exacerbates Lethal Experimental Sepsis.

Low-level lead exposure is associated with aberrant sperm quality and reproductive hormone levels in Chinese male individuals: Results from the MARHCS study low-level lead exposure is associated with aberrant sperm quality.