Armen Saghatelyan

Mice deficient for the HNK-1 sulfotransferase show alterations in synaptic efficacy and spatial learning and memory.

Recognition molecule associated carbohydrate inhibits postsynaptic GABA(B) receptors: a mechanism for homeostatic regulation of GABA release in perisomatic synapses.

Tenascin-R mediates activity-dependent recruitment of neuroblasts in the adult mouse forebrain.

Reduced GABAergic transmission and number of hippocampal perisomatic inhibitory synapses in juvenile mice deficient in the neural cell adhesion molecule L1.

Nicotinic receptors regulate the survival of newborn neurons in the adult olfactory bulb.

Local neurons play key roles in the mammalian olfactory bulb.

Inhibitory interneurons in the olfactory bulb: from development to function.

Activity-dependent adjustments of the inhibitory network in the olfactory bulb following early postnatal deprivation.

Integrating new neurons into the adult olfactory bulb: joining the network, life-death decisions, and the effects of sensory experience.

Neuronal fate determinants of adult olfactory bulb neurogenesis.

Neonatal and adult neurogenesis provide two distinct populations of newborn neurons to the mouse olfactory bulb.

Delayed onset of odor detection in neonatal mice lacking tenascin-C.

A dlx2- and pax6-dependent transcriptional code for periglomerular neuron specification in the adult olfactory bulb.

Vasculature guides migrating neuronal precursors in the adult mammalian forebrain via brain-derived neurotrophic factor signaling.

Role of blood vessels in the neuronal migration.

Interneurons produced in adulthood are required for the normal functioning of the olfactory bulb network and for the execution of selected olfactory behaviors.

De-routing neuronal precursors in the adult brain to sites of injury: role of the vasculature.

Role of sensory activity on chemospecific populations of interneurons in the adult olfactory bulb.

Newborn neurons in the adult olfactory bulb: unique properties for specific odor behavior.

Astrocytes control the development of the migration-promoting vasculature scaffold in the postnatal brain via VEGF signaling.