Maria Pia Rastaldi

Depletion of clusterin in renal diseases causing nephrotic syndrome.

Apolipoprotein E in idiopathic nephrotic syndrome and focal segmental glomerulosclerosis.

Glomerular podocytes possess the synaptic vesicle molecule Rab3A and its specific effector rabphilin-3a.

Induction of B7-1 in podocytes is associated with nephrotic syndrome.

Histopathological atlas of renal diseases--IgA nephropathy.

Histopathological atlas of renal diseases. Membranoproliferative glomerulonephritis.

Histopathological atlas of renal diseases: membranous glomerulonephritis.

Histopathological atlas of renal diseases: Acute post-streptococcal glomerulonephritis.

Functional consequences of integrin-linked kinase activation in podocyte damage.

Human renal epithelial cells produce the long pentraxin PTX3.

Histopathological atlas of renal diseases: Minimal Change disease and focal glomerulosclerosis.

Histopathological atlas of renal diseases: ANCA-associated vasculitis (first part).

Histopathological atlas of renal diseases: ANCA-associated vasculitis (Second part).

Histopathological atlas of renal diseases: light chain deposition disease.

Henoch-Schonlein nephritis.

Role of TGF-beta/GLUT1 axis in susceptibility vs resistance to diabetic glomerulopathy in the Milan rat model.

Histopathological atlas of renal diseases: diabetic nephropathy.

Glomerular podocytes contain neuron-like functional synaptic vesicles.

Glomerular activation of the lectin pathway of complement in IgA nephropathy is associated with more severe renal disease.

Renal amyloidosis (part I).

Better kidney health for Europe.

Renal amyloidosis (Part II).

Loss of the tumor suppressor Vhlh leads to upregulation of Cxcr4 and rapidly progressive glomerulonephritis in mice.

Epithelial-mesenchymal transition and its implications for the development of renal tubulointerstitial fibrosis.

Necrotizing crescentic glomerulonephritis--a conditional knockout model discloses new therapeutic challenges.

Expression of filtrin in human glomerular diseases.

Demonstration of secretory IgA in kidneys of patients with IgA nephropathy.

Epithelial-mesenchymal transition (EMT) of renal tubular cells in canine glomerulonephritis.

The role of secretory IgA and complement in IgA nephropathy.

The death ligand TRAIL in diabetic nephropathy.

The MIF receptor CD74 in diabetic podocyte injury.

Dog as model for down-expression of E-cadherin and beta-catenin in tubular epithelial cells in renal fibrosis.

Role of basic fibroblast growth factor (FGF-2) in diabetic nephropathy and mechanisms of its induction by hyperglycemia in human renal fibroblasts.

Restoration of podocyte structure and improvement of chronic renal disease in transgenic mice overexpressing renin.

Both complement and IgG fc receptors are required for development of attenuated antiglomerular basement membrane nephritis in mice.

alpha- and beta-Adducin polymorphisms affect podocyte proteins and proteinuria in rodents and decline of renal function in human IgA nephropathy.

Cannabinoid receptor 1 blockade ameliorates albuminuria in experimental diabetic nephropathy.

BASP1 promotes apoptosis in diabetic nephropathy.

A transgenic mouse model for uromodulin-associated kidney diseases shows specific tubulo-interstitial damage, urinary concentrating defect and renal failure.

The detrimental effects of IFN-α on vasculogenesis in lupus are mediated by repression of IL-1 pathways: potential role in atherogenesis and renal vascular rarefaction.

BAMBI is expressed in endothelial cells and is regulated by lysosomal/autolysosomal degradation.

Adducin- and ouabain-related gene variants predict the antihypertensive activity of rostafuroxin, part 1: experimental studies.

Post-transplant proteinuria associated with everolimus: Definition of main features with proteomics.

Albuminuria and glomerular damage in mice lacking the metabotropic glutamate receptor 1.

Role of mTOR in podocyte function and diabetic nephropathy in humans and mice.

Recent insights into vitamin D and its receptor.

Nephrin expression in adult rodent central nervous system and its interaction with glutamate receptors.

Rituximab targets podocytes in recurrent focal segmental glomerulosclerosis.

Alpha-actinin-4 and CLP36 protein deficiencies contribute to podocyte defects in multiple human glomerulopathies.

The inhibiting Fc receptor for IgG, FcγRIIB, is a modifier of autoimmune susceptibility.

Circulating urokinase receptor as a cause of focal segmental glomerulosclerosis.

Protective role of cannabinoid receptor type 2 in a mouse model of diabetic nephropathy.

[Synaptic-like signals at the filtration barrier: the role of nephrin].

Crk1/2-dependent signaling is necessary for podocyte foot process spreading in mouse models of glomerular disease.

[Hypocomplementemic tubulointerstitial nephritis in IgG4-related disease].

BAMBI regulates angiogenesis and endothelial homeostasis through modulation of alternative TGFβ signaling.

Compensatory molecular and functional mechanisms in nervous system of the Grm1(crv4) mouse lacking the mGlu1 receptor: a model for motor coordination deficits.

Fifteen years of research on nephrin: what we still need to know.

The RenTg mice: a powerful tool to study renin-dependent chronic kidney disease.

Calcium and osteoprotegerin levels predict the progression of the abdominal aortic calcifications after kidney transplantation.

Podocyte expression of membrane transporters involved in puromycin aminonucleoside-mediated injury.

Defining cell-type specificity at the transcriptional level in human disease.

Allogeneic mesenchymal stem cell infusion for the stabilization of focal segmental glomerulosclerosis.

Urinary exosomal microRNAs in incipient diabetic nephropathy.

Current indications to parathyroidectomy in CKD patients before and after renal transplantation.

The first case of IgG4-related disease in Italy.

Role of Podocyte B7-1 in Diabetic Nephropathy.

The sclerosing glomerulus in mice and man: novel insights.

Positive effects of a novel non-peptidyl low molecular weight radical scavenger in renal ischemia/reperfusion: a preliminary report.