Gary R. Whittaker

Role of the influenza virus M1 protein in nuclear export of viral ribonucleoproteins.

Dissecting virus entry via endocytosis.

Influenza virus can enter and infect cells in the absence of clathrin-mediated endocytosis.

Role of protein kinase C betaII in influenza virus entry via late endosomes.

Differential requirements of Rab5 and Rab7 for endocytosis of influenza and other enveloped viruses.

Virus nuclear import.

Rab GTPases are recruited to chlamydial inclusions in both a species-dependent and species-independent manner.

Role for influenza virus envelope cholesterol in virus entry and infection.

The ubiquitin-vacuolar protein sorting system is selectively required during entry of influenza virus into host cells.

Influenza virus entry and infection require host cell N-linked glycoprotein.

Role of clathrin-mediated endocytosis during vesicular stomatitis virus entry into host cells.

The avian coronavirus infectious bronchitis virus undergoes direct low-pH-dependent fusion activation during entry into host cells.

Bioinformatic and comparative localization of Rab proteins reveals functional insights into the uncharacterized GTPases Ypt10p and Ypt11p.

Fluorescence dequenching assays of coronavirus fusion.

Avian infectious bronchitis virus enters cells via the endocytic pathway.

Feline aminopeptidase N is not a functional receptor for avian infectious bronchitis virus.

Heparan sulfate is a selective attachment factor for the avian coronavirus infectious bronchitis virus Beaudette.

Role of the actin cytoskeleton during influenza virus internalization into polarized epithelial cells.

Molecular architecture of the bipartite fusion loops of vesicular stomatitis virus glycoprotein G, a class III viral fusion protein.

Differential role for low pH and cathepsin-mediated cleavage of the viral spike protein during entry of serotype II feline coronaviruses.

Utilization of DC-SIGN for entry of feline coronaviruses into host cells.

Activation of p38 MAPK by feline infectious peritonitis virus regulates pro-inflammatory cytokine production in primary blood-derived feline mononuclear cells.

Activation of the SARS coronavirus spike protein via sequential proteolytic cleavage at two distinct sites.

Characterization of a highly conserved domain within the severe acute respiratory syndrome coronavirus spike protein S2 domain with characteristics of a viral fusion peptide.

SARS-coronavirus spike S2 domain flanked by cysteine residues C822 and C833 is important for activation of membrane fusion.

Feline lectin activity is critical for the cellular entry of feline infectious peritonitis virus.

Elastase-mediated activation of the severe acute respiratory syndrome coronavirus spike protein at discrete sites within the S2 domain.

Modifications to the hemagglutinin cleavage site control the virulence of a neurotropic H1N1 influenza virus.

Promotion of vesicular stomatitis virus fusion by the endosome-specific phospholipid bis(monoacylglycero)phosphate (BMP).

Expression of the C-type lectins DC-SIGN or L-SIGN alters host cell susceptibility for the avian coronavirus, infectious bronchitis virus.

Characterization of a recombinant canine coronavirus with a distinct receptor-binding (S1) domain.

Cleavage activation of the human-adapted influenza virus subtypes by matriptase reveals both subtype and strain specificities.

Mechanisms of coronavirus cell entry mediated by the viral spike protein.

Influenza virus-mediated membrane fusion: determinants of hemagglutinin fusogenic activity and experimental approaches for assessing virus fusion.

Influenza virus-membrane fusion triggered by proton uncaging for single particle studies of fusion kinetics.

Acquisition of a novel eleven amino acid insertion directly N-terminal to a tetrabasic cleavage site confers intracellular cleavage of an H7N7 influenza virus hemagglutinin.

Plasmin-mediated activation of pandemic H1N1 influenza virus hemagglutinin is independent of the viral neuraminidase.

Internalization and fusion mechanism of vesicular stomatitis virus and related rhabdoviruses.

Cleavage activation of human-adapted influenza virus subtypes by kallikrein-related peptidases 5 and 12.

Mutation in spike protein cleavage site and pathogenesis of feline coronavirus.

Entry of influenza virus.

Entry of rhabdoviruses into animal cells.

Single particle assay of coronavirus membrane fusion with proteinaceous receptor-embedded supported bilayers.

Utilization of human DC-SIGN and L-SIGN for entry and infection of host cells by the New World arenavirus, Junín virus.

A novel activation mechanism of avian influenza virus H9N2 by furin.

Influenza entry pathways in polarized MDCK cells.

Inhibition of influenza virus infection and hemagglutinin cleavage by the protease inhibitor HAI-2.

Canine enteric coronaviruses: emerging viral pathogens with distinct recombinant spike proteins.

Genotypic characterization of canine coronaviruses associated with fatal canine neonatal enteritis in the United States.

Host cell entry of Middle East respiratory syndrome coronavirus after two-step, furin-mediated activation of the spike protein.

Variations in pH sensitivity, acid stability, and fusogenicity of three influenza virus H3 subtypes.

Host cell proteases: Critical determinants of coronavirus tropism and pathogenesis.

Modification of the hemagglutinin cleavage site allows indirect activation of avian influenza virus H9N2 by bacterial staphylokinase.

Human transferrin receptor triggers an alternative Tacaribe virus internalization pathway.

Recombinant M2e outer membrane vesicle vaccines protect against lethal influenza A challenge in BALB/c mice.

Fusion of Enveloped Viruses in Endosomes.

Middle East respiratory syndrome coronavirus infection is inhibited by griffithsin.

Viral fusion efficacy of specific H3N2 influenza virus reassortant combinations at single-particle level.

A camel-derived MERS-CoV with a variant spike protein cleavage site and distinct fusion activation properties.

Murine Leukemia Virus (MLV)-based Coronavirus Spike-pseudotyped Particle Production and Infection.

Safe Recombinant Outer Membrane Vesicles that Display M2e Elicit Heterologous Influenza Protection.

A peptide-based approach to evaluate the adaptability of influenza A virus to humans based on its hemagglutinin proteolytic cleavage site.

Use of AAScatterPlot tool for monitoring the evolution of the hemagglutinin cleavage site in H9 avian influenza viruses.

A single dose and long lasting vaccine against pandemic influenza through the controlled release of a heterospecies tandem M2 sequence embedded within detoxified bacterial outer membrane vesicles.

The SARS-CoV Fusion Peptide Forms an Extended Bipartite Fusion Platform that Perturbs Membrane Order in a Calcium-Dependent Manner.

Physiological and molecular triggers for SARS-CoV membrane fusion and entry into host cells.

Improving Virus Taxonomy by Recontextualizing Sequence-Based Classification with Biologically Relevant Data: the Case of the Species.

Feline coronavirus: Insights into viral pathogenesis based on the spike protein structure and function.

Characterizing replication kinetics and plaque production of type I feline infectious peritonitis virus in three feline cell lines.