Wuqiang Zhu

Overexpression of bone morphogenetic protein 10 in myocardium disrupts cardiac postnatal hypertrophic growth.

Expression of a mutant p193/CUL7 molecule confers resistance to MG132- and etoposide-induced apoptosis independent of p53 or Parc binding.

Cardiac restricted overexpression of kinase-dead mammalian target of rapamycin (mTOR) mutant impairs the mTOR-mediated signaling and cardiac function.

The CUL7 E3 ubiquitin ligase targets insulin receptor substrate 1 for ubiquitin-dependent degradation.

A mouse model for juvenile doxorubicin-induced cardiac dysfunction.

Smad7 is required for the development and function of the heart.

Acute doxorubicin cardiotoxicity is associated with p53-induced inhibition of the mammalian target of rapamycin pathway.

Cell-cycle-based strategies to drive myocardial repair.

Hypoxic preconditioning enhances the benefit of cardiac progenitor cell therapy for treatment of myocardial infarction by inducing CXCR4 expression.

Differential cardiac remodeling in preload versus afterload.

Dishevelled-associated activator of morphogenesis 1 (Daam1) is required for heart morphogenesis.

FKBP12 is a critical regulator of the heart rhythm and the cardiac voltage-gated sodium current in mice.

Granulocyte colony-stimulating factor treatment plus dipeptidylpeptidase-IV inhibition augments myocardial regeneration in mice expressing cyclin D2 in adult cardiomyocytes.

Restrictive loss of plakoglobin in cardiomyocytes leads to arrhythmogenic cardiomyopathy.

Tbx20 transcription factor is a downstream mediator for bone morphogenetic protein-10 in regulating cardiac ventricular wall development and function.

Triggered firing and atrial fibrillation in transgenic mice with selective atrial fibrosis induced by overexpression of TGF-β1.

Fkbp1a controls ventricular myocardium trabeculation and compaction by regulating endocardial Notch1 activity.

P53 inhibition exacerbates late-stage anthracycline cardiotoxicity.

Critical Roles of STAT3 in β-Adrenergic Functions in the Heart.

31P NMR 2D Mapping of Creatine Kinase Forward Flux Rate in Hearts with Postinfarction Left Ventricular Remodeling in Response to Cell Therapy.

Biomarkers for monitoring chemotherapy-induced cardiotoxicity.

Meeting Report for the 2017 National Institutes of Health National Heart, Lung, and Blood Institute Progenitor Cell Biology Consortium: Cardiovascular Bioengineering Workshop and Symposium.

Overcoming the Roadblocks to Cardiac Cell Therapy Using Tissue Engineering.

CCND2 Overexpression Enhances the Regenerative Potency of Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes: Remuscularization of Injured Ventricle.

Pathologic Stimulus Determines Lineage Commitment of Cardiac C-kit Cells.

VEGF nanoparticles repair the heart after myocardial infarction.

Large Cardiac Muscle Patches Engineered From Human Induced-Pluripotent Stem Cell-Derived Cardiac Cells Improve Recovery From Myocardial Infarction in Swine.

Spheroids of cardiomyocytes derived from human-induced pluripotent stem cells improve recovery from myocardial injury in mice.

Can We Engineer a Human Cardiac Patch for Therapy?

Regenerative Potential of Neonatal Porcine Hearts.

Y-27632 preconditioning enhances transplantation of human-induced pluripotent stem cell-derived cardiomyocytes in myocardial infarction mice.

Convergences of Life Sciences and Engineering in Understanding and Treating Heart Failure.