Nadia A. Ameen

Molecular motors and apical CFTR traffic in epithelia.

Characterization of CFTR High Expresser cells in the intestine.

Restoration of cytoskeletal and membrane tethering defects but not defects in membrane trafficking in the intestinal brush border of mice lacking both myosin Ia and myosin VI.

Identification of intestinal ion transport defects in microvillus inclusion disease.

AP2 α modulates cystic fibrosis transmembrane conductance regulator function in the human intestine.

Linaclotide activates guanylate cyclase-C/cGMP/protein kinase-II-dependent trafficking of CFTR in the intestine.

Zinc ameliorates intestinal barrier dysfunctions in shigellosis by reinstating claudin-2 and -4 on the membranes.

Glucocorticoids and myosin5b loss of function induce heightened PKA signaling in addition to membrane traffic defects.

Glucocorticoids and serum- and glucocorticoid-inducible kinase 1 are potent regulators of CFTR in the native intestine: implications for stress-induced diarrhea.

CFTR High Expresser Cells in cystic fibrosis and intestinal diseases.